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[Etiological diagnostic methods and research progress of forest encephalitis].
Zhonghua Lao Dong Wei Sheng Zhi Ye Bing Za Zhi
February 2024
Department of Occupational Poisoning, Heilongjiang Institute of Labor Hygiene and Occupational Diseases, Harbin 150028, China.
Forest encephalitis is a natural focal disease transmitted through the bite of hard ticks, and its pathogen is the tick-borne encephalitis virus from the Flaviviridae family. The mortality rate of forest encephalitis is relatively high, making laboratory testing significant in diagnosing this disease. This article elaborates on the etiological diagnostic methods and recent research progress in forest encephalitis.
View Article and Find Full Text PDFPhylogenetic analysis of tick-borne encephalitis virus strains found in an engorged tick and traveler returning from Russia.
Ticks Tick Borne Dis
May 2021
Department of Veterinary Pathobiology, College of Veterinary Medicine and Biomedical Sciences, Texas A&M University, College Station, TX, 77843-4467, USA. Electronic address:
Although travel-related tick-borne encephalitis (TBE) cases have been increasingly registered worldwide, very few published case studies are available to date. The present report describes a travel-related TBE case and provides genotypic characterization of two viral isolates. Laboratory diagnostics were based on complement fixation test and virus isolation.
View Article and Find Full Text PDFReactivation of Histoplasmosis Disseminated into the Central Nervous System Presenting as a Stroke.
Cureus
December 2019
Internal Medicine, Citrus Memorial Hospital, Inverness, USA.
Histoplasmosis is one of the most prevalent endemic mycosis in the United States. Patients with a previous history of histoplasmosis have a risk of reinfection in the future. Individuals with impaired immunity and those who have massive re-exposure to H.
View Article and Find Full Text PDFThe clinical features, underlying immunology, and treatment of autoantibody-mediated movement disorders.
Mov Disord
September 2018
Oxford Autoimmune Neurology Group, Nuffield Department of Clinical Neurosciences, University of Oxford, Oxford, UK.
An increasing number of movement disorders are associated with autoantibodies. Many of these autoantibodies target the extracellular domain of neuronal surface proteins and associate with highly specific phenotypes, suggesting they have pathogenic potential. Below, we describe the phenotypes associated with some of these commoner autoantibody-mediated movement disorders, and outline increasingly well-established mechanisms of autoantibody pathogenicity which include antigen downregulation and complement fixation.
View Article and Find Full Text PDFAutoantibody-mediated diseases of the CNS: Structure, dysfunction and therapy.
Neuropharmacology
April 2018
Autoimmune Neurology Group, Nuffield Department of Clinical Neurosciences, University of Oxford, Oxford, OX3 9DS, UK. Electronic address:
The field of neuronal autoantibody associated diseases of the central nervous system has expanded dramatically in the last few years. The range of identified neuronal and glial antibody targets has led to the accurate classification of a number of syndromes which each associate with characteristic clinical features. These diseases are especially important due to their frequent response to immunotherapies.
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