AI Article Synopsis
- The study examines the relationship between lactate levels and arterial base deficit in diagnosing traumatic brain injury (TBI) in patients with isolated head injuries.
- Only 17% of the 131 patients studied were found to have TBI, but the differences in arterial base deficit and lactate levels between those with and without TBI were not significant.
- The conclusion highlights that both arterial base deficit and lactate levels are ineffective in identifying TBI in cases of isolated head trauma.
Article Abstract
Background: Increase in lactate (LAC) within the central nervous system after head trauma is an established marker of traumatic brain injury (TBI).
Objective: To investigate the utility of arterial base deficit (BD) and LAC in identifying TBI in patients with isolated head injury (IHI).
Materials And Methods: TBI was defined as Glasgow Coma Scale < or =8, head Abbreviated Injury Severity Score >2 or brain haematoma on CT scan. Patients were divided into two groups: IHI with and without TBI. Data were reported as means (SDs). 131 patients with IHI were studied (mean (SD) age 39 (19) years, 78% male).
Results: 17% of the patients sustained TBI. The mean differences for arterial BD (0.65 mmol/l, 95% CI -0.8 to 2.1) and LAC (0.34 mmol/l, 95% CI -0.7 to 1.4) in patients with and without TBI were not significant. Analysis of receiver operating characteristic curves confirmed that arterial BD and LAC were unable to detect TBI in patients with IHI.
Conclusion: Arterial BD and LAC are poor predictors of TBI in isolated head trauma.
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| http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2658477 | PMC |
| http://dx.doi.org/10.1136/emj.2006.044578 | DOI Listing |
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