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http://dx.doi.org/10.1136/jnnp.2005.083618 | DOI Listing |
Neurorehabil Neural Repair
April 2023
Spinal Cord Injury Center, Balgrist University Hospital, Zurich, Switzerland.
Background: Sufficient and timely spinal cord decompression is a critical surgical objective for neurological recovery in spinal cord injury (SCI). Residual cord compression may be associated with disturbed cerebrospinal fluid pressure (CSFP) dynamics.
Objectives: This study aims to assess whether intrathecal CSFP dynamics in SCI following surgical decompression are feasible and safe, and to explore the diagnostic utility.
Phlebology
March 2015
Department of Neurology, S. Pio Hospital, Vasto (CH), Italy.
Objective: To evaluate the utility of a transcranial brain photoplethysmography parameter as a potential marker for patients with multiple sclerosis.
Methods: We investigated 38 patients affected by multiple sclerosis, according to the revised McDonald criteria (12 males and 26 females, mean age 41.1 ± 8.
J Neurol Neurosurg Psychiatry
June 2006
Department of Neurology, Hull Royal Infirmary, UK.
Headache
February 2003
Department of Neurology, University Hospital Charité, Humboldt University, Berlin, Germany.
Background: Cerebral venous distension is thought by some to serve as a source of migraine pain. Previous investigators have tried to modify pain intensity by induction of additional venous congestion via compression of both internal jugular veins (Queckenstedt's maneuver). The magnitude of blood flow within the internal jugular veins depends markedly on body position, and inconsistencies in positioning may have influenced their results.
View Article and Find Full Text PDFJ Neurol Neurosurg Psychiatry
August 1998
Department of Neurology, Glostrup Hospital, University of Copenhagen, Denmark.
Evidence for the involvement of the cranial arterial system in migraine is plentiful, but it is unclear whether the cranial venous system may be involved in the mechanism of migraine pain. Venules are the preferentially involved vessels in the neurogenic inflammation animal model of migraine. The cranial and cerebral veins and sinuses are pain sensitive and receive sensory innervation from the trigeminal nerve.
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