Experiments were conducted on albino rats to study the changes of the amplitude and latency of maxima tract and synaptic components of the dorsal surface potential before and after spinal cord injury at the level of Th9. Dorsal surface potentials were recorded from L4 after stimulation in C2. The amplitude of tract and synaptic component maxima of the short latency potential were found to be considerably reduced after the injury. The latency of both components increased after the injury. The changes of the amplitude or latency of potentials evoked from different stimulated points of the cord were analysed. The possible mechanism of the revealed changes is discussed.

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