Background: Both disorder-specific cognitions and unconditional core beliefs have been associated with eating-disordered behaviours. This study examines whether these beliefs might provide competing or complementary explanations of those behaviours.
Method: The participants were 151 women with eating disorders. Each woman completed two self-report measures-the Eating Disorder Examination Questionnaire (measuring disorder-specific cognitions and reported behavioural frequency) and the Young Schema Questionnaire-Short version (measuring unconditional core beliefs). Objective height and weight were measured to give body mass index (BMI). Regression analyses were used to compare additive, mediator and moderator models of the cognition-behaviour link.
Results: BMI and reported frequency of vomiting were best explained by models where the impact of disorder-specific cognitions was moderated by unhealthy core beliefs, but where neither form of belief had an independent effect. In contrast, the frequency of reported objective binge-eating was best explained by an additive effect of the two forms of cognition.
Discussion: The findings indicate that both disorder-specific cognitions and unconditional core beliefs are necessary to explain the development and maintenance of disordered eating behaviours. This conclusion suggests that cognitive-behavioural approaches might be more effective if they address both levels of cognition. However, prospective research is required to confirm the causal hypothesis based on the present cross-sectional data.
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http://dx.doi.org/10.1016/j.eatbeh.2005.09.001 | DOI Listing |
Behav Res Ther
December 2024
Rogers Behavioral Health, Research Center, Oconomowoc WI & Philadelphia PA, 34700 Valley Rd, Oconomowoc, WI, 53066, USA. Electronic address:
The presentation of avoidant/restrictive food intake disorder (ARFID) characterized by fear of aversive consequences of eating (fear-ARFID) has both phenomenological and mechanistic similarities to panic disorder. In this narrative review, we propose a shared model of the pathogenesis of the two disorders, centered on interoceptive sensitivity as the key maintenance mechanism. We review the evidence that fear-ARFID, which involves restrictive eating motivated by a desire to avoid aversive events (e.
View Article and Find Full Text PDFActa Psychiatr Scand
December 2024
VIRTU Research Group, Mental Health Center Copenhagen, Copenhagen University Hospital - Mental Health Services, CPH, Denmark.
Background: The increasing number of studies of immersive virtual reality (VR) interventions for mental disorders call for an examination of the current level of evidence on their effectiveness. The findings may guide scalability and contribute to the advancement and optimization of immersive VR-based interventions for mental disorders.
Methods: A systematic literature search across four databases screened 2443 studies.
Transl Psychiatry
December 2024
State Key Laboratory of Cognitive Neuroscience and Learning, Beijing Normal University, Beijing, 100875, China.
Neurodynamic models that simulate how micro-level alterations propagate upward to impact macroscopic neural circuits and overall brain function may offer valuable insights into the pathological mechanisms of schizophrenia (SCZ). In this study, we integrated a neurodynamic model with the classical Contrastive Variational Autoencoder (CVAE) to extract and evaluate macro-scale SCZ-specific features, including subject-level, region-level parameters, and time-varying states. Firstly, we demonstrated the robust fitting of the model within our multi-site dataset.
View Article and Find Full Text PDFCereb Cortex
December 2024
State Key Laboratory of Primate Biomedical Research, Institute of Primate Translational Medicine, Kunming University of Science and Technology, No. 727 Jingming South Road, Chenggong District, Kunming 650500, China.
Postpartum depression and postpartum depression with anxiety, which are highly prevalent and debilitating disorders, become a growing public concern. The high overlap on the symptomatic and neurobiological levels led to ongoing debates about their diagnostic and neurobiological uniqueness. Delineating the shared and disorder-specific intrinsic functional connectivities and their causal interactions is fundamental to precision diagnosis and treatment.
View Article and Find Full Text PDFPeerJ Comput Sci
November 2024
Laboratory for Social & Cognitive Informatics, National Research University Higher School of Economics, St. Petersburg, Russia.
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