According to the National Wild Turkey Federation, there has been a 450% increase in the number of licensed wild turkey hunters since 1973. Unfortunately, there is a paucity of research articles focusing specifically on turkey hunting. Most human dimensions of wildlife research have focused on either deer or waterfowl hunting. In this study, dimensions of hunting quality, days a field, and harvest were examined among a sample of 739 Virginia fall turkey hunters. Quality of a hunting season, rather than a specific hunt, was examined. Principal components analysis (PCA) revealed five domains of attributes of a high-quality turkey-hunting season: wildlife interaction, education, being away, social interaction, and hunting skills. The domains of hunting quality revealed by the PCA are similar to domains found previously for spring turkey hunters as well as other types of hunters. Also examined were hunter perceptions of overall hunting quality, the number of days spent hunting turkeys during the fall season, and harvest success. For comparative purposes and agency needs, hunters were segmented into three categories based on days spent hunting: low frequency (1 to 2 days), medium frequency (3 to 4 days), and high frequency (> or =5 days). The influence of these variables to respondents' perceptions of overall quality was examined using a series of regression models. Variables found to be significant predictors of quality were wildlife interaction and harvest success. Hunters who killed a turkey reported a significantly higher quality season than those who did not kill a turkey.
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Int J Pharm
December 2024
Hacettepe University, Faculty of Medicine, Department of Histology and Embryology, 06100, Sıhhiye, Ankara, Turkey; METU MEMS Center, 06530, Ankara, Turkey. Electronic address:
Rapid progressing non-small cell lung adenocarcinoma (NSCLC) decreases treatment success. Cannabinoids emerge as drug candidates for NSCLC due to their anti-tumoral capabilities. We previously reported the controlled release of Arachidonylcyclopropylamide (ACPA) selectively targeting cannabinoid 1 (CB1) receptor in NSCLC cells in vitro.
View Article and Find Full Text PDFJAMA Netw Open
January 2025
Division of Cancer Genetics and Prevention, Dana-Farber Cancer Institute, Boston, Massachusetts.
Importance: CHEK2 pathogenic and likely pathogenic variants (PVs) are common, and low-risk (LR) variants, p.I157T, p.S428F, and p.
View Article and Find Full Text PDFTurk J Med Sci
December 2024
Department of General Surgery, Faculty of Medicine, Giresun University, Giresun, Turkiye.
Background/aim: To investigate the association between the rs2267437, rs5751129 and rs132770 polymorphisms of the gene, which plays a role in repairing DNA double-strand breaks, and the risk of papillary thyroid carcinoma (PTC).
Materials And Methods: The study included 150 patients who had been diagnosed with PTC and 204 healthy controls. Genotyping of the SNPs was performed using polymerase chain reaction-restriction fragment length polymorphism (PCR-RFLP).
Parasitol Res
December 2024
Department of Parasitology, Faculty of Veterinary Medicine, Ondokuz Mayıs University, Samsun, Turkey.
The superfamily Ascaridoidea are parasitic nematodes in vertebrates, including birds and humans. In order to investigate the presence and distribution of these parasitic nematodes in birds acting as the definitive host, 157 birds of 64 bird species belonging to 16 orders were collected and necropsied in the Kızılırmak Delta area in the Bafra district of Samsun province. The parasites collected were fixed in 70% ethyl alcohol and identified under a light microscope, and morphologically important regions were photographed for identification.
View Article and Find Full Text PDFbioRxiv
December 2024
Istanbul Medipol University, School of Engineering and Natural Sciences, Department of Biomedical Engineering, 34810, Istanbul, Turkey.
G protein-coupled receptor (GPCR) signaling is terminated by arrestin binding to a phosphorylated receptor. Binding propensity has been shown to be modulated by stabilizing the pre-activated state of arrestin through point mutations or C-tail truncation. Here, we hypothesize that pre-activated rotated states can be stabilized by small molecules, and this can promote binding to phosphorylation-deficient receptors, which underly a variety of human disorders.
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