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Mitochondrial Dysfunction in PCOS: Insights into Reproductive Organ Pathophysiology.
Int J Mol Sci
August 2023
Department of Pediatrics, Division of Neonatology, Sanford School of Medicine, University of South Dakota, 1400 W. 22nd St., Sioux Falls, SD 57105, USA.
Article Synopsis
- *This review collects data from humans and animals to explore how mitochondrial activity affects reproductive organs and overall health in individuals with PCOS.
- *It aims to clarify the role of mitochondria in not just current reproductive health but also in the possible inheritance of PCOS and related metabolic conditions.
Polycystic ovary syndrome (PCOS) and hyperandrogenism: the role of a new natural association.
Minerva Ginecol
October 2015
Department of Molecular and Developmental Medicine, University of Siena, Siena, Italy -
Aim: Polycystic ovary syndrome (PCOS) affects 5-10% of women of childbearing age and manifests itself through oligomenorrhea, anovulation, hirsutism, micro-polycystic ovaries. Insulin resistance is a characteristic of PCOS patients and is more pronounced in obese patients. Insulin resistance and consequent hyperinsulinemia are related to many aspects of the syndrome such as hyperandrogenism, reproductive disorders, acne and hirsutism.
View Article and Find Full Text PDFLaparoscopic diagnosis and treatment of pelvic benign multicystic mesothelioma associated with high CA19.9 serum concentration.
J Minim Invasive Gynecol
July 2010
Department of Gynecology, Obstetrics, and Neonatology, University Medical School of Bari, Bari, Italy.
We report a case of benign multicystic mesothelioma in a 20-year-old woman referred because of amenorrhea. She underwent pelvic transabdominal ultrasound, which disclosed a micropolycystic appearance of the ovaries and a fluid collection in the pouch of Douglas. Tumor serum markers revealed an increase in CA19.
View Article and Find Full Text PDF[Obesity and altered arterial structure in young women with micropolycystic ovary syndrome].
Rev Bras Ginecol Obstet
July 2009
Departamento de Ginecologia e Obstetrícia, Universidade de São Paulo, Ribeirão Preto, SP, Brasil.
Purpose: to compare echographical cardiovascular risk factors between obese and non-obese patients with micropolycystic ovarian syndrome (MPOS).
Methods: in this transversal study, 30 obese (Body Mass Index, BMI>30 kg/m(2)) and 60 non-obese (BMI<30 kg/m(2)) MPOS patients, aging between 18 and 35 years old, were included. The following variables were measured: flow-mediated dilatation (FMD) of the brachial artery, thickness of the intima-media of the carotid artery (IMT), anthropometric data, systolic arterial pressure (SAP) and diastolic arterial pressure (DAP).
Expanding the phenotype of 22q11 deletion syndrome: the MURCS association.
Clin Dysmorphol
January 2008
Medical Genetics, Department of Molecular Biology Department of Internal Medicine, Endocrine-Metabolic Sciences and Biochemistry, University of Siena, Siena Department of Endocrinology, University of Pisa, Pisa Medical Genetics, Hospital of reggio Calabria, Bianchi-Melacrino-Morelli, Reggio Calabria, Italy.
The MURCS association [Müllerian Duct aplasia or hypoplasia (M), unilateral renal agenesis (UR) and cervicothoracic somite dysplasia (CS)] manifests itself as Müllerian Duct aplasia or hypoplasia, unilateral renal agenesis and cervicothoracic somite dysplasia. We report on a 22-year-old woman with bicornuate uterus, right renal agenesis, C2-C3 vertebral fusion (MURCS association) and 22q11.2 deletion.
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