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Induction of mRNA for tumor necrosis factor-alpha and interleukin-1 beta in mice brain, spleen and liver in an animal model of Shigella-related seizures.
Isr Med Assoc J
February 2000
Department of Pediatrics A, Schneider Children's Medical Center of Israel, Petah Tiqva, Israel.
Background: The pathogenesis of neurological symptoms, the most common extraintestinal complication of childhood shigellosis, is unclear. To elucidate the mechanisms involved, we developed an animal model and demonstrated that TNF alpha and IL-1 beta play a role.
Objectives: To determine whether TNF alpha and IL-1 beta genes are expressed in the brain following peripheral administration of Shigella dysenteriae 60R.
Acinetobacter meningitis: four nosocomial cases.
J Formos Med Assoc
March 1999
Department of Neurology, Chang Gung Memorial Hospital-Kaohsiung, Taiwan.
We report the clinical features and therapeutic outcomes of four patients with multiantibiotic-resistant Acinetobacter meningitis. There were three males and one female, aged from 17 to 49 years. Three of them had suffered from head injuries with skull fractures, and the other suffered from an intracerebral hemorrhage and underwent a craniotomy.
View Article and Find Full Text PDFAztreonam in the treatment of gram-negative bacterial meningitis.
Rev Infect Dis
August 1991
Department of Medicine, Helene Fuld Medical Center, Trenton, New Jersey 08638.
Aztreonam was administered to 122 patients with presumptive or confirmed gram-negative bacillary meningitis in an open, multinational study. The antibiotic was administered at a dosage of 1-2 g to adults, 50 mg/kg to children greater than 2 years old, and 30 mg/kg to infants three or four times daily. Seventy-seven patients had microbiologically confirmed gram-negative meningitis due to an aztreonam-susceptible organism and received aztreonam for at least 48 hours.
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