AI Article Synopsis
- The case describes a 43-year-old man diagnosed with Miller Fisher syndrome (MFS) who experienced rapid onset symptoms including ptosis, eye muscle paralysis, and severe ataxia.
- Initial MRI scans revealed lesions in the cauda equina, leading to the diagnosis and subsequent immunotherapy treatment, which improved some symptoms but not all.
- A follow-up MRI five months later showed lesions in the spinal posterior column, suggesting they were affecting the posterior nerve roots and contributing to the persistent severe ataxia.
Article Abstract
The site of lesions causing ataxia in Miller Fisher syndrome (MFS) remains in dispute. A 43-year-old man manifested rapidly progressive left-sided ptosis, bilateral abducens palsy, areflexia, and severe ataxia. Initial MR imaging showed confined lesions of the cauda equina with gadolinium enhancement. A diagnosis of MFS was made, and the patient underwent immunotherapy. His ophthalmoplegia disappeared, but other symptoms remained. Five months after onset, MR imaging disclosed lesions confined to the spinal posterior column, which were considered to result from involvement of posterior nerve roots of the cauda equina and to be responsible for his remaining severe ataxia.
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| http://www.ncbi.nlm.nih.gov/pmc/articles/PMC7975592 | PMC |
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