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Impact of hypokalemia on Brugada syndrome: case report unveiling mechanisms beyond QT interval prolongation.
Egypt Heart J
October 2024
Department of Cardiology and Vascular Medicine, Faculty Medicine, Gorontalo State University,, Jalan Jend, Sudirman No.6, Dulalowo Kecamatan Kota Tengah Kota, Gorontalo, 96128, Indonesia.
Article Synopsis
- Brugada syndrome (BrS) increases the risk of sudden death due to dangerous heart rhythms and is characterized by specific ECG changes, notably ST-segment elevation in right precordial leads.
- A case involving a 51-year-old man with low potassium levels (hypokalemia) revealed that while treatment normalized potassium, the Brugada pattern on ECG remained unchanged, and his corrected QT interval was shorter during hypokalemia.
- This case suggests that hypokalemia can reveal the Brugada type-1 pattern without changing it, indicating that further research is needed to explore how hypokalemia could influence ventricular arrhythmias beyond just QT interval changes.
Predicting ventricular arrhythmia inducibility in ajmaline-induced Brugada type I pattern: Validation of the dST-Tiso interval.
J Cardiovasc Electrophysiol
September 2024
Maria Cecilia Hospital, GVM Care & Research, Cotignola, Ravenna, Italy.
Introduction: The dST-Tiso is a newly proposed electrocardiographic (ECG) marker during Brugada (BrS) type I pattern, that predicts the likelihood of ventricular arrhythmia (VA) inducibility in patients with ajmaline-induced pattern. The objective of this study was to validate the effectiveness of this criterion using an independent data set.
Methods: Consecutive patients exhibiting a BrS type I ECG pattern following ajmaline administration underwent programmed ventricular stimulation (PVS).
Long-term follow-up of patients with Brugada syndrome: Foremost risk factors associated with overall arrhythmic events.
Medicine (Baltimore)
May 2024
Department of Cardiology, Private Clinic, Ankara, Turkey.
Brugada syndrome (BS) is characterized by ST segment elevation in right precordial leads (V1-V3), ventricular tachycardia (VT), ventricular fibrillation (VF), and sudden cardiac death (SCD) in individuals without structural heart disease. The aim of this study is to contribute to the controversial issue of finding the most valuable marker that can predict poor prognosis during follow-up in patients with a diagnosis of BS. A total of 68 patients diagnosed with BS or had Brugada-type ECG change between January 1997 and July 2012 at the Department of Cardiology of Başkent University Faculty of Medicine, Ankara, Turkey, were included in this cohort study.
View Article and Find Full Text PDFSevere hypothyroidism as a trigger for Brugada-type ECG abnormalities: a case report and literature review.
Arch Endocrinol Metab
February 2024
Divisione di Endocrinologia, Diabetologia e Metabolismo, Dipartimento di Scienze Mediche, Università di Torino, Torino, Italia.
Article Synopsis
- Brugada syndrome (BrS) is a genetic disorder that can lead to dangerous heart rhythms and sudden death in people with normal heart structures, with hypothyroidism being a potential triggering factor.
- A 33-year-old man with severe hypothyroidism exhibited a Brugada type 1 ECG pattern, and the relationship between severe thyroid dysfunction and ECG alterations is still not fully understood.
- Treatment with levothyroxine, even at low doses, led to a quick improvement in the patient's ECG readings, suggesting that hypothyroidism may contribute to Brugada syndrome symptoms.
More than 30 years of Brugada syndrome: a critical appraisal of achievements and open issues.
Herzschrittmacherther Elektrophysiol
March 2024
Heart Center Bremen, Electrophysiology Bremen, Klinikum Links der Weser, Bremen, Germany.
Over the last three decades, what is referred to as Brugada syndrome (BrS) has developed from a clinical observation of initially a few cases of sudden cardiac death (SCD) in the absence of structural heart disease with ECG signs of "atypical right bundle brunch block" to a predominantly electrocardiographic, and to a lesser extent genetic, diagnosis. Today, BrS is diagnosed in patients without overt structural heart disease and a spontaneous Brugada type 1 ECG pattern regardless of symptoms. The diagnosis of BrS is less clear in those with an only transient or drug-induced type 1 Brugada pattern, but should be considered in the presence of an arrhythmic syncope, family history of BrS, or family history of sudden death.
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