We assessed the contractility of the contralateral wall on 2-dimensional echocardiography in 50 patients with an initial ST-elevation acute myocardial infarction who underwent coronary angiography. Compensatory hyperkinesis, which we defined as a fractional thickening of >/=60% in the contralateral wall, was a strong predictor of single-vessel coronary artery disease, with a positive predictive value of 85%.
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http://dx.doi.org/10.1016/j.amjcard.2003.09.038 | DOI Listing |
J Vet Cardiol
April 2023
Clinic for Small Animal Medicine, Department of Veterinary Medicine, Ludwig-Maximilians-Universität Munich, Veterinärstrasse 13, 80469 Munich, Germany. Electronic address:
Introduction/objectives: Pulmonic stenosis (PS) is one of the most common congenital heart diseases in dogs leading to right ventricular (RV) pressure overload, myocardial remodeling, and potential RV dysfunction. Our objectives were to investigate the extent of RV systolic dysfunction in canine PS and to examine the immediate influence of balloon valvuloplasty (BV) on systolic function.
Animals, Materials And Methods: This prospective study evaluated 72 dogs with PS and 86 healthy dogs.
Zh Nevrol Psikhiatr Im S S Korsakova
January 2022
Children's City Polyclinic No. 110, Moscow, Russia.
Objective: To determine the electromyographic patterns of motor tics in children.
Material And Methods: 130 patients were examined, which were divided into 3 groups: the 1st group included 21 patients with motor tics; the 2nd group included 87 patients with motor-vocal tics; the 3rd group included 22 patients with Tourette's syndrome (ST). The control group consisted of 35 healthy children.
Mol Brain
March 2021
Graduate School of Pharmacy, Ritsumeikan University, Kusatsu, Shiga, Japan.
The 15q13.3 microdeletion syndrome is a genetic disorder characterized by a wide spectrum of psychiatric disorders that is caused by the deletion of a region containing 7 genes on chromosome 15 (MTMR10, FAN1, TRPM1, MIR211, KLF13, OTUD7A, and CHRNA7). The contribution of each gene in this syndrome has been studied using mutant mouse models, but no single mouse model recapitulates the whole spectrum of human 15q13.
View Article and Find Full Text PDFEchocardiography
March 2021
Division of Cardiovascular Medicine, Department of Internal Medicine, Kasai City Hospital, Hyogo, Japan.
The mechanism for dynamic left ventricular outflow tract obstruction (LVOTO) after acute coronary syndromes (ACS) is thought to be apical infarction with compensatory hyperkinesia of the residual normally perfused basal segments of the myocardium. However, herein, we report a patient with ACS and dynamic LVOTO (peak gradient of 250 mm Hg at rest) that could not be secondary to apical akinesia. We propose a potential alternative mechanism leading to dynamic LVOTO in ACS, namely, the interplay between sigmoid septum, basal hyperkinesis, and outflow tract narrowing induced by afterload reduction due to acute myocardial ischemia itself.
View Article and Find Full Text PDFWorld J Clin Cases
December 2020
Department of Cardiology, The First Affiliated Hospital, College of Medicine, Zhejiang University, Hangzhou 310000, Zhejiang Province, China.
Background: Takotsubo cardiomyopathy (TTC), a syndrome of acute left ventricular (LV) dysfunction, is characterized by transitory hypokinesis of LV apices with compensatory hyperkinesis of the LV basal region. The symptoms of TTC mimic acute myocardial infarction, without significant coronary stenoses on coronary angiography. Echocardiogram plays a key role in the diagnosis and prognosis of TTC.
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