Severity: Warning
Message: file_get_contents(https://...@pubfacts.com&api_key=b8daa3ad693db53b1410957c26c9a51b4908&a=1): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests
Filename: helpers/my_audit_helper.php
Line Number: 176
Backtrace:
File: /var/www/html/application/helpers/my_audit_helper.php
Line: 176
Function: file_get_contents
File: /var/www/html/application/helpers/my_audit_helper.php
Line: 250
Function: simplexml_load_file_from_url
File: /var/www/html/application/helpers/my_audit_helper.php
Line: 1034
Function: getPubMedXML
File: /var/www/html/application/helpers/my_audit_helper.php
Line: 3152
Function: GetPubMedArticleOutput_2016
File: /var/www/html/application/controllers/Detail.php
Line: 575
Function: pubMedSearch_Global
File: /var/www/html/application/controllers/Detail.php
Line: 489
Function: pubMedGetRelatedKeyword
File: /var/www/html/index.php
Line: 316
Function: require_once
Although everybody agrees that each individual's blood pressure variance is under substantial genetic control, its quantitative estimates vary from 35 to 70%, according to the different experimental designs. The problem is that, in spite of such substantial contribution, the discovery of the "genes that cause hypertension" has been quite discouraging. This is indeed caused by the multifactorial nature of the disease and no improvement is to be expected without dissecting hypertension in its major pathogenetic pathways. As a paradigm of such a dissection, we present the data on adducin system, that proved to be relevant for a common form of salt sensitive in humans and the genetic hypertension of MHS rats.
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