30-day changes in enteral enzymes were followed up in test and control growing white rats. Test animals received protein- and calorie-deficient feeding. It was established that retardation of growth was more marked in protein than in calorie deficiency.
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Ann Saudi Med
December 2024
From the Faculty of Medicine, University of Pristina, Medical Statistics and Informatics, Kosovska Mitrovica, Serbia.
Background: Postoperative complications of colorectal cancer surgery contribute to increased morbidity and mortality in patients.
Objectives: Investigate the role of biochemical markers in serum and peritoneal fluid in the development of postoperative complications in patients with enteral anastomosis.
Design: Prospective.
Early Hum Dev
January 2025
Universidade Federal do Rio Grande do Sul, Faculdade de Medicina, Programa de Pós-Graduação em Ciências Médicas: Endocrinologia, Porto Alegre, Rio Grande do Sul, Brazil; Hospital de Clínicas de Porto Alegre, Serviço de Neonatologia, Porto Alegre, Rio Grande do Sul, Brazil; Hospital de Clínicas de Porto Alegre, Serviço de Nutrologia, Porto Alegre, Rio Grande do Sul, Brazil. Electronic address:
Background: Metabolic bone disease (MBD) of newborns (NB) is characterized by tissue reduction and inadequate postnatal bone mineralization with clinical, laboratory, and/or radiologic repercussions between the third and twelfth weeks of postnatal life, which, in its most severe forms, can lead to a growth deficit and fractures. The aim of our study is to evaluate the incidence of MBD in premature patients receiving parenteral nutrition for >15 days in the neonatal intensive care unit (NICU) and assess their clinical and laboratory characteristics.
Methods: Single-center retrospective cohort study.
Eur J Pediatr
November 2024
Faculty of Medicine, Mansoura University, Mansoura, Egypt.
Clin Nutr ESPEN
December 2024
Nutrition-Diabetes Department, University Hospital of Montpellier, Montpellier, France; UMR 1302, Institute Desbrest of Epidemiology and Public Health, Univ Montpellier, INSERM, University Hospital of Montpellier, Montpellier, France.
Background & Aims: Glycogenosis type III is a rare autosomal recessive disease caused by a mutation in the AGL gene that results in a deficiency of the glycogen debranching enzyme. This deficiency impairs fasting tolerance and leads to hypoglycemia. While the symptomatology tends to improve with age, pregnancy can trigger a recurrence of hypoglycemia due to increased carbohydrate requirements.
View Article and Find Full Text PDFNutrients
October 2024
Division of Surgical Oncology, Department of Surgery, University of Louisville, Louisville, KY 40202, USA.
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