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Greater mechanical temporal summation of pain in Latinx-Americans and the role of adverse life experiences.
Pain Rep
September 2020
Department of Psychological and Brain Sciences, Texas A&M University, College Station, TX, USA.
Introduction: Adverse life experiences disproportionately impact Latinx-Americans and are related to greater chronic pain rates. However, little is known about how adversities interact with central pain mechanisms for the development of later pain among Latinx-Americans.
Objectives: The current study examined the relationship between adverse life experiences (eg, trauma and ethnic discrimination) and correlates (eg, social status) with mechanical temporal summation of pain (a proxy measure of central sensitization) between pain-free U.
Role of neuroendocrine and neuroimmune mechanisms in chronic inflammatory rheumatic diseases--the 10-year update.
Semin Arthritis Rheum
December 2013
Laboratory of Experimental Rheumatology and Neuroendocrino-Immunology, Division of Rheumatology, Department of Internal Medicine I, University Hospital, Regensburg, Germany. Electronic address:
Background: Neuroendocrine immunology in musculoskeletal diseases is an emerging scientific field. It deals with the aspects of efferent neuronal and neurohormonal bearing on the peripheral immune and musculoskeletal systems. This review aims to add new information that appeared since 2001.
View Article and Find Full Text PDFSkeletal muscle α-actin diseases (actinopathies): pathology and mechanisms.
Acta Neuropathol
January 2013
Centre for Medical Research, The University of Western Australia, Crawley, Australia.
Mutations in the skeletal muscle α-actin gene (ACTA1) cause a range of congenital myopathies characterised by muscle weakness and specific skeletal muscle structural lesions. Actin accumulations, nemaline and intranuclear bodies, fibre-type disproportion, cores, caps, dystrophic features and zebra bodies have all been seen in biopsies from patients with ACTA1 disease, with patients frequently presenting with multiple pathologies. Therefore increasingly it is considered that these entities may represent a continuum of structural abnormalities arising due to ACTA1 mutations.
View Article and Find Full Text PDF[Cytokines in psychoneuroendocrine immunological context of nonspecific musculoskeletal pain].
Schmerz
August 2012
Klinik für Orthopädie und Unfallchirurgie, Vulpius Klinik GmbH, Bad Rappenau, Deutschland.
Cytokines are coordinators of immune homeostasis. Evidence for the participation of cytokines in neurogenic inflammation, peripheral and central sensitization and hyperalgesia as well as for induction of inflammatory immune responses by pain-related catastrophizing is well documented. A disproportion of proinflammatory and anti-inflammatory cytokines is known to be a contributory cause of pain and pain behavior.
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