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Portal vein stenting in recurrent or locally advanced peri-hilar cholangiocarcinoma.
Eur J Surg Oncol
March 2024
Department of Surgery, Dentistry, Gynecology and Pediatrics, Division of General and Hepatobiliary Surgery, University of Verona, G.B. Rossi University Hospital, P. le L.A. Scuro 10, 37134, Verona, Italy. Electronic address:
Background: Recurrent or locally advanced peri-hilar cholangiocarcinoma (PHCC) usually involves the portal vein (PV) leading to significant stenosis. With disease progression, clinical symptoms such as ascites, bleeding, and hepatic insufficiency are usually observed. Little is know about the benefit of PV stenting in relieving the symptoms associated to portal hypertension and allowing anticancer therapies.
View Article and Find Full Text PDFA new variant of Abernethy malformation treated by transhepatic interventional closure: a case report.
BMC Gastroenterol
February 2022
Department of Pediatric Cardiology, University Children's Hospital, University of Tübingen, Hoppe-Seyler-Str. 1, 72076, Tübingen, Germany.
Background: Congenital portosystemic shunts (CPSS) are rare vascular malformations and can be classified into extrahepatic and intrahepatic shunts. Extrahepatic CPSS, also termed Abernethy malformations are associated with severe long-term complications including portopulmonary hypertension, liver atrophy, hyperammoniemia and hepatic encephalopathy. We report a hitherto undescribed variant of Abernethy malformation requiring an innovative approach for interventional treatment.
View Article and Find Full Text PDF[Kinetics of nitrogenous metabolites in the kidney during chronic tetrachloromethane hepatitis].
Patol Fiziol Eksp Ter
November 2014
The kinetics of ammonia, glutamine, and urea in the kidney has been studied in experiments on 203 white rats (females) at the end of chronic tetrachloromethane (CCl4) exposure (65 days) and within 14 days after cessation of CCl4. It was found that on the 65th day of CCl4 administration the arterial hyperammoniemia is formed, which lasts for 14 days after the abolition of the toxin. This is accompanied by an increased excretion of ammonia in the urine and an increase in its concentration in the blood of renal veins, which does not prevent its accumulation in renal tissue.
View Article and Find Full Text PDF[Hyperammoniemia due to portal-systemic shunt developed after initiation of hemodialysis in a chronic renal failure patient].
Nihon Naika Gakkai Zasshi
August 2004
Jikei University, School of Medicine, Department of Internal Medicine, Division of Kidney and Hypertension, Tokyo.
Surgical treatment for an extrahepatic portosystemic shunt: a case report.
Hepatogastroenterology
September 2004
Department of Surgery, Wakayama Rosai Hospital, Wakayama, Japan.
An extrahepatic portosystemic shunt that has neither liver cirrhosis nor portal hypertension is rare. A 60-year-old Japanese woman who had been suffering chronic liver disease and anemia with mild disorientation was admitted to investigate general fatigue with dizziness and disorientation. The laboratory data revealed mild pancytopenia and liver dysfunction including hyperammoniemia, an increased Indocyanine Green 15-min retention rate, and a decreased Fischer's ratio.
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