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Structural and molecular reorganization of cardiomyocytes in transposition of the main vessels.
Bull Exp Biol Med
September 2010
E. N. Meshalkin Institute of Circulatory Pathology, Rosmedtechnologies, Novosibirsk, Russia.
Studies of myocardial autopsy specimens from infants (0-12 months) with transposition of the main vessels showed the formation of a complex of compensatory adaptive, degenerative, and destructive changes, manifesting at a tissue level in cardiomyocyte heterogeneity and formation of cardiosclerosis zones. Cardiac myosin synthesis was replaced by synthesis of skeletal myosin, which was detected at the molecular level. Clinically it manifested in the progress of heart failure.
View Article and Find Full Text PDFMicrocirculatory bed of the epi- and pericardium, parietal pleura, bulbar conjunctiva is studied in congenital heart defects. The alterations of the microvessels were similar in all above organs. The following conclusions are made: a) the reduction of microvessels manifests as capillary failure of the myocardium resulting in cardiomyocyte destruction and development of cardiosclerosis, b) myocardial alterations result in heart failure and enhancement of systemic microhemocirculation, c) irreversible pathological processes can be prevented by early surgery only.
View Article and Find Full Text PDFDifferences in the diagnose panorama in primary health care in Dalby, Sweden and Spili, Crete.
Scand J Soc Med
March 1993
Department of Family and Social Medicine, Crete University, Greece.
We have compared the use of primary health care and the diagnoses at visits to doctors in the Spili Health Centre (SHC) in Crete and the Dalby Health Centre (DHC) in Sweden. In DHC more patients per 1000 population visited the doctors than in SHC. This was so regardless of age-group and sex, in fact more or less regardless of diagnosis.
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