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Cerebellar restricted diffusion in Wernicke's encephalopathy.
J Neurosci Rural Pract
November 2022
Department of Neurology, Johns Hopkins University, Baltimore, Maryland, United States.
A 50-year-old woman presented to an outside hospital for acute onset bilateral lower limb pain. She was diagnosed with aortoiliac stenosis and underwent stent placement. Post-procedure, she was noted to have altered mental status, truncal ataxia, neck titubation, and incomplete external ophthalmoplegia.
View Article and Find Full Text PDFEvidence of enteric angiopathy and neuromuscular hypoxia in patients with mitochondrial neurogastrointestinal encephalomyopathy.
Am J Physiol Gastrointest Liver Physiol
May 2021
Department of Morphology, Surgery and Experimental Medicine, University of Ferrara, Ferrara, Italy.
Mitochondrial neurogastrointestinal encephalomyopathy (MNGIE) is a rare autosomal recessive disease caused by thymidine phosphorylase (TP) enzyme defect. As gastrointestinal changes do not revert in patients undergone TP replacement therapy, one can postulate that other unexplored mechanisms contribute to MNGIE pathophysiology. Hence, we focused on the local TP angiogenic potential that has never been considered in MNGIE.
View Article and Find Full Text PDFCharacterisation of tau in the human and rodent enteric nervous system under physiological conditions and in tauopathy.
Acta Neuropathol Commun
July 2018
Inserm, U1235, 1 rue Gaston Veil, F-44035, Nantes, France.
Tau is normally a highly soluble phosphoprotein found predominantly in neurons. Six different isoforms of tau are expressed in the adult human CNS. Under pathological conditions, phosphorylated tau aggregates are a defining feature of neurodegenerative disorders called tauopathies.
View Article and Find Full Text PDFAnti-GQ1b antibody syndrome presenting as acute isolated bilateral ophthalmoplegia: Report on two patients and review of the literature.
Eur J Paediatr Neurol
May 2016
Department of Paediatric Neurology, Centre Hospitalier Universitaire Saint-Pierre (U.L.B.), Rue Haute 322, 1000 Brussels, Belgium; Department of Paediatric Neurology, Hôpital Universitaire des Enfants Reine Fabiola (U.L.B.), Avenue Crocq 15, 1020 Brussels, Belgium.
Background: Miller Fisher syndrome (MFS) is an acute polyradiculoneuritis regarded as an uncommon clinical variant of Guillain-Barré syndrome (GBS). MFS is characterized by the acute onset of the clinical triad of ophthalmoplegia, cereballar ataxia and areflexia. Atypical forms of MFS presenting as isolated ophthalmoplegia without ataxia have been rarely described, mostly in adults.
View Article and Find Full Text PDF[Wernicke's encephalopathy and Caine criteria. Report of six cases].
Rev Med Inst Mex Seguro Soc
April 2015
Servicio de Neurología, Hospital de Especialidades, Centro Médico Nacional La Raza, Instituto Mexicano del Seguro Social, Distrito Federal, México.
Background: Wernicke's encephalopathy is an acute and reversible neurologic disorder due to deficiency of thiamin. Chronic alcoholism was the main cause in the past; currently, there are many other situations which favour this condition: prolonged intravenous feeding, hyperemesis gravidarum, anorexia nervosa, regional enteritis, malabsorption syndrome, hemodialysis, peritoneal dialysis, and abdominal surgery.
Clinical Case: We report six patients, three male and three female, who had in common total parenteral nutrition over two months, secondary to abdominal surgery complications and restriction to enteral nutrition.
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