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Ureteral motility.
Acta Physiol Hung
December 2009
Department of Urology and Clinical Experimental Research Department, Semmelweis University, Budapest, Hungary.
The pyeloureteral function is to transport urine from the kidneys into the ureter toward the urinary bladder for storage until micturition. A set of mechanisms collaborates to achieve this purpose: the basic process regulating ureteral peristalsis is myogenic, initiated by active pacemaker cells located in the renal pelvis. Great emphasis has been given to hydrodynamic factors, such as urine flow rate in determining the size and pattern of urine boluses which, in turn, affect the mechanical aspects of peristaltic rhythm, rate, amplitude, and baseline pressure.
View Article and Find Full Text PDF[Ionic stretch-dependent channels in the urogenital tract. Pharmaco-physiology of tocolysis].
Gynakol Geburtshilfliche Rundsch
April 1994
Urolog. Klinik, Krankenhaus Barmherzigen Brüder, München.
[Pharmacophysiology of inotropic agents].
Minerva Anestesiol
October 1992
Istituto Pluridisciplinare di Anestesiologia, Rianimazione e Terapia Intensiva, Università Degli Studi di Messina.
Advances in our understanding of the pathophysiology of cardiac-ventricular failure and the pharmacophysiology of adrenergic receptors have greatly improved the short-term therapy of the low-output, hypotensive-hypoperfused, cardiac failure states. Agents are now specifically selected for these conditions on the basis of their predominant effects on the heart (positive inotropy) and the peripheral vasculature (vasodilatation or vasoconstriction). With the addition of dobutamine therapy, the pharmacophysiologic spectrum now includes norepinephrine (predominant vasopressor), dopamine (combined vasopressor-positive inotrope), dobutamine (predominant positive inotrope), and isoproterenol (combined positive inotrope-vasodilator).
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