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http://dx.doi.org/10.1046/j.1365-4362.2003.01723_2.x | DOI Listing |
Int J Surg Case Rep
January 2024
Department of Pharmacy Practice, Karavali College of Pharmacy, Vamanjoor (post), Mangalore 575028, Karnataka, India.
Introduction: Zosteriform Lichen Planus represents a relatively uncommon variant of LP. It is characterized by a distinctive distribution following Blaschko's line and involving multiple dermatomes, setting it apart as a unique manifestation. There have been several cases of cutaneous LP reported, but relatively few of them presented as zosteriform LP.
View Article and Find Full Text PDFJ Yeungnam Med Sci
October 2023
Department of Dermatology, Keimyung University School of Medicine, Daegu, Korea.
Porokeratosis ptychotropica is an uncommon form of porokeratosis, which was initially described in 1995. It is clinically characterized by symmetrical reddish to brown-colored hyperkeratotic, verrucous, or psoriasiform plaques on the perianal and gluteal regions. The lesions tend to integrate and expand centrally, with small peripheral satellite lesions.
View Article and Find Full Text PDFBr J Dermatol
October 2015
Department of Dermatology, Salamanca University Hospital, Paseo San Vicente, 88-182, 37007, Salamanca, Spain.
Cutaneous graft-versus-host disease (GVHD) is a frequent complication of allogeneic bone marrow transplant and haematopoietic cell transplantation, but it is rarely presented as a Wolf's isotopic response. We report a patient who developed chronic lichenoid GVHD following the dermatomes previously affected by varicella zoster virus (VZV) infection. Nineteen months later, the same patient suffered from reactivation of GVHD at the injection site of an influenza vaccination.
View Article and Find Full Text PDFIndian J Dermatol
November 2013
Visakha Institute of Skin and Allergy, Marripalem, Visakhapatnam, Andhra Pradesh, India.
Background: Segmental vitiligo (SV) and generalized vitiligo (GV) are perceived to evolve by different mechanisms, the former with unspecified neural mechanisms and the latter by melanocyte specific autoimmune mechanisms. However, the two diverse mechanisms are difficult to reconcile in cases of "mixed vitiligo". To test the possibility of a common pathogenesis, we reviewed clinical and histopathological features of SV and GV.
View Article and Find Full Text PDFInt J Dermatol
July 2003
Department of Dermatology, Medical Faculty, Ankara University, Ankara, Turkey.
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