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An Introduction to Spiral Steroids.
Int J Mol Sci
August 2022
IOMA LLC, Belmont, CA 94002-3321, USA.
In addition to classical steroids, which have cholesterol as a precursor, there are steroids with 7-dehydrocholesterol as a precursor. This review describes the identification of these steroids, their biosynthesis, and some aspects of their function. There are three classes of these compounds, distinguished by the number of their carbon atoms, 23, 24, and 25.
View Article and Find Full Text PDFInvolvement of reactive oxygen species in a feed-forward mechanism of Na/K-ATPase-mediated signaling transduction.
J Biol Chem
November 2013
Department of Pharmacology, Physiology and Toxicology, JCE School of Medicine at Marshall University, Huntington, West Virginia 25755. Electronic address:
Cardiotonic steroids (such as ouabain) signaling through Na/K-ATPase regulate sodium reabsorption in the renal proximal tubule. We report here that reactive oxygen species are required to initiate ouabain-stimulated Na/K-ATPase·c-Src signaling. Pretreatment with the antioxidant N-acetyl-L-cysteine prevented ouabain-stimulated Na/K-ATPase·c-Src signaling, protein carbonylation, redistribution of Na/K-ATPase and sodium/proton exchanger isoform 3, and inhibition of active transepithelial (22)Na(+) transport.
View Article and Find Full Text PDFPostnatal maturation of the dog stria vascularis-- an immunohistochemical study.
Anat Rec A Discov Mol Cell Evol Biol
January 2003
Laboratory of Anatomy and Embryology, Veterinary Anatomy, Free University of Brussels, Brussels, Belgium.
The lateral wall of the dog cochlear duct was investigated by classical staining and immunohistochemistry for NaK/ATPase beta2 isoform, cytokeratins (Cks), vimentin, nestin, and S100A6 during the postnatal cochlear maturation, i.e., from birth to postnatal day 110.
View Article and Find Full Text PDFGinkgo biloba extract (EGb 761) protects Na,K-ATPase isoenzymes during cerebral ischemia.
Cell Mol Biol (Noisy-le-grand)
September 2002
Laboratoire de Recherche Cardiologique, Université de la Méditerranée, Faculté de Médecine, IFR Jean-Roche, Marseille, France.
Disturbances of Na,K-ATPase activity are implicated in the pathophysiology of cerebral ischemia. Previous experiments have shown that EGb 761 protects NaK-ATPase activity against one hour of cerebral ischemia. In the brain however, the 3 isoenzymes responsible for Na,K-ATPase activity may be differentially affected by various times of ischemia.
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