Nonreinforced exposure to a nontarget stimulus that was followed by nonreinforced exposure to a target/nontarget simultaneous compound stimulus resulted in enhanced latent inhibition of the target. Conditioning was slower after this treatment than after nonreinforced exposure to the target stimulus alone (Experiment 1). However, a salient auditory stimulus presented immediately after the compound in the second phase reduced levels of latent inhibition, relative to the enhanced latent inhibition produced when no such extracompound stimulus was presented (Experiments 2 and 3). This effect was not noted if the salient auditory cue was presented 10 sec after the termination of the compound stimulus (Experiment 4). In Experiment 5, there was no disruption of simple latent inhibition produced by a salient stimulus. These results are consistent with enhanced latent inhibition's being produced by the formation of within-compound associations, which are disrupted by the salient extracompound stimuli.
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Adv Sci (Weinh)
January 2025
School of Chemistry and Chemical Engineering, Frontiers Science Center for Transformative Molecules, Shanghai Jiao Tong University, Shanghai, 200240, China.
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The Wistar Institute, Philadelphia, Pennsylvania, USA.
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Biomedical Research Institute of Southern California, Oceanside, CA, United States.
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School and Hospital of Stomatology, Wenzhou Medical University, Wenzhou, China; Department of Endodontics, School and Hospital of Stomatology, Wenzhou Medical University, Wenzhou, China. Electronic address:
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Division of Psychology and Language Sciences, UCL, London WC1H 0AP, UK. Electronic address:
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