Facilitation among morphologically related words generally is impervious to the prefixed or suffixed structure of primes and targets. A notable exception arises, however, when both primes and targets are suffixed. More specifically, when primes are auditory and targets are visual, facilitation for a suffixed target (e.g., payment) is absent when it follows a prime (e.g., payable) that is morphologically related and suffixed (Marslen-Wilson, Tyler, Waksler, & Older, 1994). To account for null facilitation (viz., the "suffix-suffix" effect), Marslen-Wilson and his colleagues posit inhibitory links between suffixes of morphological relatives. The present study assesses the generality of the "suffix-suffix" effect. When morphological facilitation is assessed relative to an orthographically related baseline, suffixed primes facilitate derivationally as well as inflectionally related morphological targets when primes are visual as well as auditory in both the lexical decision and naming tasks. The present findings call into question lexical models that posit inhibitory links between suffixes of morphological relatives.
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http://dx.doi.org/10.1006/brln.2001.2504 | DOI Listing |
Open Forum Infect Dis
January 2025
Department of Medicine, Division of Infectious Diseases, University of Pittsburgh and UPMC, Pittsburgh, Pennsylvania, USA.
Background: Improved diagnostic testing (DT) of infections may optimize outcomes for solid organ transplant recipients (SOTR), but a comprehensive analysis is lacking.
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J Neuroinflammation
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Department of Neurology, Division of Neuroimmunology, School of Medicine, Johns Hopkins University, Baltimore, MD, 21287, USA.
Chronic innate immune activation in the central nervous system (CNS) significantly contributes to neurodegeneration in progressive multiple sclerosis (MS). Using multiple experimental autoimmune encephalomyelitis (EAE) models, we discovered that NLRX1 protects neurons in the anterior visual pathway from inflammatory neurodegeneration. We quantified retinal ganglion cell (RGC) density and optic nerve axonal degeneration, gliosis, and T-cell infiltration in Nlrx1 and wild-type (WT) EAE mice and found increased RGC loss and axonal injury in Nlrx1 mice compared to WT mice in both active immunization EAE and spontaneous opticospinal encephalomyelitis (OSE) models.
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January 2025
Istituto Oncologico Veneto IOV-IRCCS, Padua, Italy.
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Singapore Immunology Network (SIgN), A*STAR, Singapore, Singapore.
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View Article and Find Full Text PDFBio Protoc
January 2025
International Institute of Food Innovation Co., Ltd., Nanchang University, Nanchang, China.
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