Introduction: Wall stress or wall tension is a conception derived from physics (Laplace's law) and represents the systolic force or work per surface unit. It is the systolic force made by myocardial tissues. Stress increase indicates enlargement of the left ventricle or increase of intracavitary pressure.
Material And Methods: This investigation included 170 subjects; control group consisted of 50 patients (pts) with normal coronary angiographic finding without valvular anomalies and the examination group included 120 pts with coronary disease. Transthoracic echocardiography was performed in the left lateral position using computerized Hewlett Packard SONOS 1000 apparatus. Invasive hemodynamic procedure was performed using GENERAL ELETRICS CGR 300. Meridional and equatorial systolic and diastolic stress were calculated according to Grossman formula.
Results: The meridional end-diastolic equatorial stress was 18.55 +/- 12.12 dyn/cm2 x 10(3) in the control group, while in coronary patients it was 28.15 +/- 13.42 dyn/cm2 x 10(3). In healthy persons the meridional end-systolic stress established by echocardiography was 190.37 +/- 23.15 dyn/cm2 x 10(3), while in coronary patients 203.82 +/- 17.88 dyn/cm2 x 10(3). End-diastolic equatorial stress was 34.32 +/- 17.18 dyn/cm2 x 10(3) in the control group and 46.13 +/- 17.82 dyn/cm2 x 10(3) in coronary patients. Systolic equatorial stress in the control group was 357.42 +/- 32.15 dyn/cm2 x 10(3) and in coronary patients 385.34 +/- 35.72 dyn/cm2 x 10(3). The same parameters determined by invasive hemodynamic procedure were slightly higher, but without statistical significance in relation to the values determined by echocardiography (P > 0.05).
Conclusion: Values of equatorial and particularly meridional stress were higher in coronary patients in relations to healthy persons, but without significant difference. The correlation coefficients of all investigated parameters established by noninvasive 2D echocardiography and invasive hemodynamic procedure were in one domain of medium high and high values. Meridional stress increases in coronary patients, equatorial in hypertensive patients or valvular anomalies with severe myocardial hypertrophy. In regard to high correlation between these two techniques, echocardiography may be considered a highly reliable method in evaluation of wall tension.
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http://dx.doi.org/10.2298/mpns0204125v | DOI Listing |
Eur Surg Res
July 2017
Department of Cardiovascular and Thoracic Surgery, Hokkaido University Graduate School of Medicine, Sapporo, Japan.
Background: The indications of left-ventricular plication (LVP) are controversial, although several studies have reported favorable outcomes in heart failure patients. The aim of this study was to assess left-ventricular (LV) wall stress and myocardial remodeling after LVP in a rat model of myocardial infarction (MI).
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Langmuir
April 2006
Department of Chemical and Biochemical Engineering, Rutgers University, 98 Brett Road, Piscataway New Jersey 08854, USA.
The linear and nonlinear rheology of viscoelastic mixed anionic-zwitterionic surfactant solutions has been systematically investigated. In the linear viscoelastic regime, these systems display nearly Maxwellian behavior with a unique relaxation time, tau0, and a characteristic elastic plateau modulus, G0. Linear rheological data were used to calculate the repitation and breaking times of the micelles, tau(rep) and tau(b), respectively.
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May 2006
First Department of Surgery, Hospital Lainz, Vienna, Austria.
Clinical in vitro endothelialization has been shown to increase the patency of synthetic vascular grafts. The shear stress resistance of the cultured autologous endothelium represents a crucial cornerstone of the concept. We investigated whether an enrichment of the precoating matrix with adhesion sites can augment endothelial cell attachment.
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Med Pregl
August 2002
Institut za kardiovaskularne bolesti, 21204 Sremska Kamenica, Institutski put bb.
Introduction: Wall stress or wall tension is a conception derived from physics (Laplace's law) and represents the systolic force or work per surface unit. It is the systolic force made by myocardial tissues. Stress increase indicates enlargement of the left ventricle or increase of intracavitary pressure.
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