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http://dx.doi.org/10.1103/PhysRevLett.88.209603 | DOI Listing |
Forensic Sci Med Pathol
September 2023
School of Automotive Engineering, Harbin Institute of Technology, Weihai, Shandong, China.
Aortic injuries are the second leading cause of death after head injuries due to traffic accidents, and strain-induced injuries are becoming increasingly prominent. The quantitative study of aortic strain injury allows for a rapid assessment of the degree of aortic injury after an accident and timely diagnosis of the pathology of aortic injury. It is more reliable than diagnosis based on clinical symptoms alone and it is faster than diagnosis based on imaging.
View Article and Find Full Text PDFBiol Psychiatry
October 2018
Department of Neurology, Baylor College of Medicine, and the Parkinson's Disease Research, Education, and Clinical Center, Veterans Health Administration, Houston, Texas. Electronic address:
Nat Mater
September 2017
Department of Physics and Center for 2-Dimensional and Layered Materials, the Pennsylvania State University, University Park, Pennsylvania 16802-6300, USA.
The magnetoelectric (ME) effect, the phenomenon of inducing magnetization by application of an electric field or vice versa, holds great promise for magnetic sensing and switching applications. Studies of the ME effect have so far focused on the control of the electron spin degree of freedom (DOF) in materials such as multiferroics and conventional semiconductors. Here, we report a new form of the ME effect based on the valley DOF in two-dimensional Dirac materials.
View Article and Find Full Text PDFJ Clin Invest
July 2007
Cardiovascular Division, Department of Medicine, Brigham and Women's Hospital, Harvard Medical School, Boston, MA 02139, USA.
Muscular dystrophies are often caused by mutations in cytoskeletal proteins that render cells more susceptible to strain-induced injury in mechanically active tissues such as skeletal or cardiac muscle. In this issue of the JCI, Han et al. report that dysferlin participates in membrane resealing in cardiomyocytes and that exercise results in increased membrane damage and disturbed cardiac function in dysferlin-deficient mice (see the related article beginning on page 1805).
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