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Drug-induced liver injury is a frequent cause of acute liver failure. It may cause clinical manifestations ranging from simple alteration of the common liver function tests until more severe manifestations including encephalopathy, coagulopathy, and in many cases progressive multi-organ dysfunction. The condition, therefore, may be associated with higher morbidity and mortality as well as higher consumption of economic resources.
View Article and Find Full Text PDF[Concentration of leptin in patients on hemodialysis and peritoneal dialysis].
Pol Arch Med Wewn
June 1999
Klinika Nefrologii i Chorób Wewnetrznych AM w Białymstoku.
Leptin, the obese gene (ob) product, is a plasma protein synthesized exclusively in adipocytes and plays an important role in regulation of food intake and energy expenditure. The aim of the study was to compare serum leptin concentration in hemodialyzed (HD) patients and in patients undergoing continuous ambulatory peritoneal dialysis (CAPD), and to assess the relationship between leptinemia and parameters of nutritional state and inflammatory markers. We studied two group of patients with end-stage renal failure: 52 hemodialyzed individuals aged 24-74 years and 19 peritoneally dialyzed patients aged 20-70 years.
View Article and Find Full Text PDF[Peritoneojugular shunt in the treatment of idiopathic ascites in the hemodialyzed patient: a clinical case].
Chir Ital
September 1996
Istituto di Clinica Chirurgica Generale, Università degli Studi, Chieti.
The case of an uremic patient receiving dialytic treatment, who also had an hydiopatic ascites, probably due to peritoneal hyper-waterproofing, is described. Peritoneo-venous shunt was positioned with good result. Generally, similar clinical situations such a therapy is as valid as other commonly used methods.
View Article and Find Full Text PDFTreatment of a patient with end-stage renal disease, severe iron overload and ascites by weekly phlebotomy combined with recombinant human erythropoietin.
Nephron
August 1990
Department of Medicine, Kawasaki Medical School, Okayama, Japan.
A 41-year-old hemodialyzed woman developed ascites and was found to have secondary iron overload. The dose of administered iron was approximately 11-12 g, and her serum ferritin level was 15,000 ng/ml (15,000 micrograms/l). There were no signs of congestive heart failure, fluid overload, or liver cirrhosis.
View Article and Find Full Text PDFLiver examination performed in seven patients who had renal failure related to light chain deposits demonstrated in all cases the presence of liver light chain deposits. In all of our patients clinical renal involvement antedated the liver disease. The portal areas and the Disse spaces contained a granular material which strongly reacted with antilight chain antiserum (kappa or gamma).
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