Echolucent or predominantly echolucent femoral plaques predict early restenosis after eversion carotid endarterectomy.

Objective: Although the association between vulnerable lesions and cardiovascular events is well established, little is known about their relationship to postsurgery restenosis. To address this issue, we initiated a prospective, nonrandomized study to examine the femoral plaques on both sides in patients who were undergoing eversion carotid endarterectomy (CEA) and were longitudinally followed-up for early restenosis development.

Methods: The final analysis enrolled 321 patients (189 women) with a median age of 67.

[Arterial thrombolysis in the lower extremity in vascular surgical practice].

The acute arterial thrombosis of the lower limb is dangerous not only for the limb, but it can cause life-threatening situation as well. That is why it is important to use different therapeutical modalities as the catheter guided intraarterial thrombolysis, which can reduce the surgical risks. All the drugs used for lysis now days are plasminogen activators.

[Congenital arteriovenous malformation complicated with brachial artery false aneurysm].

Congenital arteriovenous malformations in the upper extremity, mainly in the hand are rare. In this patient an arteriovenous fistula of the hand caused arterial dilatation of the upper limb, and blunt injury of brachial artery resulted in an extreme large false aneurysm. This situation, which threatened with rupture and peripheral embolisation was treated by resection of the false aneurysm with end-to-end direct anastomosis.

Impairment of blood-brain barrier integrity during carotid surgery as assessed by serum S-100B protein concentrations.

During carotid endarterectomy (CEA) the internal carotid artery is cross-clamped for a period of several minutes. This maneuver may cause cerebral hypoperfusion and/or impairment of the blood-brain barrier (BBB) even in cases where clinical signs are absent. The aim of the present study was to examine whether such alterations could be detected by monitoring the cerebral marker S-100B protein concentrations during and after CEA in the serum.