, a crucial target of miR-410, functions as a potential oncogene in osteosarcoma.

Purpose: Mounting evidence highlights the essential role of in tumor initiation and malignant progression in several cancers; however, the function of in osteosarcoma (OS) remains unknown. In this study, we aim to investigate the role of and reveal its regulation by deregulated miRNAs in OS.

Materials And Methods: The expression profiles of were examined by immunohistochemistry, Western blotting, and qRT-PCR.

promotes cell invasion in gastric cancer by activating the NF-κB signaling pathway.

() overexpression has been reported to be involved in the carcinogenesis and progression of many malignant tumors. However, the role of in the progression of gastric cancer (GC) remains unclear. In this study, we explored whether and how regulates proinflammatory signaling to promote GC cell invasion.

MiR-128b is down-regulated in gastric cancer and negatively regulates tumour cell viability by targeting PDK1/Akt/NF-κB axis.

Gastric cancer (GC) is the fourth most prevalent type of cancer worldwide, which is usually caused by the interaction between environmental and genetic factors, or epigenetic aspects. Referring to the non-coding RNAs, miR-128b has been reported to be associated with many tumour cases, and exerts distinct functions in different types of cancers. However, the function of miR-128b in GC onset and progression largely remains unknown.