Publications by authors named "Zhouqing Wang"

Epilepsy is characterized by spontaneous seizures. Changes in the expression of the connexins (Cxs) have been reported to be involved in epileptogenesis. It has previously been shown that the transient receptor potential vanilloid 4 (TRPV4) plays an important role in the modulation of neuronal excitability, and that application of a TRPV4 antagonist blocks hyperthermia-induced seizures.

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Activation of transient receptor potential vanilloid 4 (TRPV4) induces neuronal injury. TRPV4 activation enhances inflammatory response and promotes the proinflammatory cytokine release in various types of tissue and cells. Hyperneuroinflammation contributes to neuronal damage in epilepsy.

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Background/aims: Glycine plays an important role in regulating hippocampal inhibitory/ excitatory neurotransmission through activating glycine receptors (GlyRs) and acting as a co-agonist of N-methyl-d-aspartate-type glutamate receptors. Activation of transient receptor potential vanilloid 4 (TRPV4) is reported to inhibit hippocampal A-type γ-aminobutyric acid receptor, a ligand-gated chloride ion channel. GlyRs are also ligand-gated chloride ion channels and this paper aimed to explore whether activation of TRPV4 could modulate GlyRs.

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We investigate the surface vector plasmonic lattice solitons (PLSs) in semi-infinite graphene-pair arrays (GPAs). The surface vector PLSs are composed of two components which are associated with different band gaps. Both components undergo mutual self-trapping at the boundary of the semi-infinite structure when the self-focusing nonlinearity of graphene and the light diffraction reach a balance.

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Neurite growth is an important process for the adult hippocampal neurogenesis which is regulated by a specific range of the intracellular free Ca concentration ([Ca]). Transient receptor potential vanilloid 4 (TRPV4) is a calcium-permeable channel and activation of it causes an increase in [Ca]. We recently reported that TRPV4 activation promotes the proliferation of stem cells in the adult hippocampal dentate gyrus (DG).

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We investigate the nonlinear supermodes of surface plasmon polaritons in graphene multilayers with arbitrary number of graphene layers. Apart from the symmetric and anti-symmetric supermodes which exist in linear multilayer graphene waveguides, more asymmetric supermodes emerge in the nonlinear counterparts as the field symmetry is broken. The number of asymmetric supermodes relies largely on the layer number of graphene.

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We investigate the vector plasmonic lattice solitons (PLSs) in nonlinear graphene-pair arrays (GPAs) consisting of periodically arranged double graphene sheets, which are spatially separated. There are two dispersion bands for the Bloch modes in the array due to the coupling of surface plasmon polaritons (SPPs) between the graphene pairs. The vector PLSs composed of two components originate from the nonlinear interaction of Bloch modes in different bands.

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We investigate the plasmonic lattice solitons (PLSs) in nonlinear graphene sheet arrays (GSAs) composed of spatially separated graphene sheets embedded in dielectric. Both the nonlinearities of graphene and dielectric are considered. The self-focusing PLSs at the Brillouin zone edges can be yielded by balancing the normal diffraction of surface plasmon polaritons (SPPs) via either the nonlinear effect of graphene or self-focusing dielectric.

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