A critical review of microbial profiles in black and odorous waters.

Black and odorous waters (BOWs) are a serious environmental problem frequently reported over the past few decades. Microorganisms are identified as implementors of the black and odorous phenomenon, which play a crucial role in the decomposition and transformation of pollutants within the BOWs. However, the information on the role of microorganisms in BOWs remains elusive.

Physics-driven untrained neural network for vortex beam compensation in adaptive optics aided underwater wireless optical communications.

Orbital angular momentum (OAM) multiplexing is emerging as a critical technique for achieving high data capacity in underwater wireless optical communications (UWOC). Nonetheless, wavefront distortions induced by underwater turbulence compromise the orthogonality of OAM modes. In this paper, we introduce a physics-driven untrained learning approach for adaptive optics that operates independently of extensive amplitude datasets.

Occurrence of AG-5 Causing Root Rot on in Northwestern China.

Astragalus mongholicus is a perennial Chinese medicinal herb in the family Leguminosae widely cultivated in China. In September 2023, A. mongholicus plants in a field in Weiyuan County, Gansu Province, showed symptoms of circular or irregular brown, sunken and necrotic lesions, multiple lesions coalesced, and brown longitudinal cracks in the roots.

Synaptotagmin-1 attenuates myocardial programmed necrosis and ischemia/reperfusion injury through the mitochondrial pathway.

Programmed necrosis/necroptosis greatly contributes to the pathogenesis of cardiac disorders including myocardial infarction, ischemia/reperfusion (I/R) injury and heart failure. However, the fundamental mechanism underlying myocardial necroptosis, especially the mitochondria-dependent death pathway, is poorly understood. Synaptotagmin-1 (Syt1), a Ca sensor, is originally identified in nervous system and mediates synchronous neurotransmitter release.

A New target of ischemic ventricular arrhythmias-ITFG2.

ITFG2 is an intracellular protein known to modulate the immune response of T-cells. Our previous investigation revealed that ITFG2 specifically targets ATP5b to regulate ATP energy metabolism and maintain mitochondrial function, thereby protecting the heart from ischemic injury. However, the role of ITFG2 in ischemic ventricular arrhythmias and its underlying mechanisms have not been previously reported.