[Development and research progress of suture button fixation Latarjet procedure].

Objective: To review the development and research progress of suture button fixation Latarjet procedure.

Methods: A comprehensive literature review was conducted to summarize the development and related modified techniques of the suture button fixation Latarjet procedure.

Results: Since the Latarjet procedure was first introduced by French scholar Latarjet in 1954, it has undergone three key transformations, resulting in suture button fixation Latarjet procedure, which has shown satisfactory outcomes in treatment of recurrent shoulder dislocation.

Fabrication of hierarchical porous ethyl cellulose fibrous membrane by electro-centrifugal spinning for drug delivery systems with excellent integrated properties.

Drug delivery systems (DDSs) based on micro-and nano- fibrous membrane have been developed for decades, in which great attention has been focused on achieving controlled drug release. However, the study on the integrated performance of these drug-loaded membranes in the use of in-vitro drug delivery dressing is lacking, as clinical medication also needs consideration from the perspectives of wound safety and patient convenience. Herein, a trilayered hierarchical porous ethyl cellulose (EC) fibrous membrane based DDS (EC-DDS) was developed by electro-centrifugal spinning.

Melting centrifugally spun ultrafine poly butylene adipate--terephthalate (PBAT) fiber and hydrophilic modification.

This paper demonstrates that melt centrifugal spinning could be used to effectively fabricate degradable poly (butylene adipate--terephthalate) (PBAT) fibers with uniform fiber diameter. The hydrophobic PBAT fibers were modified into hydrophilic fibers using the hyperbranched polyesters (HBP) with three-dimensional molecular chain structures and a large number of functional groups at the chain ends. The structures and properties of the obtained fibers were characterized with SEM, XRD, DSC, contact angle, and tensile strength analyses.

HIMF deletion ameliorates acute myocardial ischemic injury by promoting macrophage transformation to reparative subtype.

Appropriately manipulating macrophage M1/M2 phenotypic transition is a promising therapeutic strategy for tissue repair after myocardial infarction (MI). Here we showed that gene ablation of hypoxia-induced mitogenic factor (HIMF) in mice (Himf and HIMF;Lyz2-Cre) attenuated M1 macrophage-dominated inflammatory response and promoted M2 macrophage accumulation in infarcted hearts. This in turn reduced myocardial infarct size and improved cardiac function after MI.