Effects of salt stress on interspecific competition between an invasive alien plant and three native species.

Biological invasions and soil salinization have become increasingly severe environmental problems under global change due to sea-level rise and poor soil management. Invasive species can often outcompete native species, but few studies focus on whether invasive alien species are always superior competitors under increasing stressors. We grew an invasive grass species, L.

Temporal transcriptome profiling reveals candidate genes involved in cold acclimation of Camellia japonica (Naidong).

Cold is a common problem that limits the distribution of Camellia. Camellia japonica (Naidong) is the northernmost species of camellia in China, which is a Tertiary remnant species that can adapt to large changes in temperature. An analysis of the transcriptional response of C.

[Ecophysiological responses of Camellia japonica (Naidong) to different light and water conditions].

Camellia japonica (Naidong), a Tertiary relict species with abundant morphological characteristics and special genetic characteristics, is the northernmost distributed population of C. japonica. The seedlings of Naidong were subjected to two light regimes (65%, 15% of full sunlight, respectively) and three water supply regimes (75%, 50% and 25% of field capacity, respectively).

[Adaptability of Camellia sasanqua leaf morphology during natural changes in temperature.].

In this study, the adaptability of leaf shape, structure and stomata characteristics of Camellia sasanqua to temperature variation in natural process was investigated by field morphological observation, conventional paraffin section and scanning electron microscopy methods. The results showed that, as the temperature decreased, the leaf color changed from green to dark green, toge-ther with white and purple spots on the leaves. The relative conductivity and the percentage of wi-thered leaves increased initially and then decreased during this period.

The checkpoint 1 kinase inhibitor LY2603618 induces cell cycle arrest, DNA damage response and autophagy in cancer cells.

Chemotherapy- or radiotherapy-induced DNA damage activates the Chk1-dependent DNA damage response (DDR) and cell cycle checkpoints to facilitate cell survival. Numerous attempts have been made to identify specific Chk1 inhibitors to enhance the efficiency of chemotherapy or radiotherapy. In this study, we investigated the molecular mechanisms underlying the antitumor activity of LY2603618, a potent and selective small molecule inhibitor of Chk1 protein kinase, in human lung cancer cells.