Phytotoxicity and nutrition behavior of process water obtained from the hydrothermal carbonization of poultry litter and its effect on lettuce germination and growth.

Nine hydrothermal carbonization process waters (PWs) of poultry litter were prepared at 180, 220, and 260 °C for 1, 4, and 8 h, respectively. They were characterized with pH, EC (electric conductivity), DOC (dissolved organic carbon), TN (total nitrogen), NH-N, NO-N, etc. After diluted according to TN, the PWs were supplied as liquid nitrogen fertilizers and their phytotoxic and nutrition effects on lettuce germination and growth were studied.

Baicalin prevents cadmium induced hepatic cytotoxicity, oxidative stress and histomorphometric alterations.

Therapeutic potential of baicalin was evaluated against Cd-induced hepatic cytotoxicity and oxidative stress. Exposure to Cd (cadmium chloride) in Chang liver cell culture produced cytotoxicity in terms of increase in cell growth inhibition rate, alanine aminotransferase, lactate dehydrogenase, and cellular lipid peroxidation, which was significantly mitigated by baicalin in a concentration dependent manner. Acute exposure to Cd (6.

Pyridoxine mitigates cadmium induced hepatic cytotoxicity and oxidative stress.

Therapeutic potential of pyridoxine (vit B6) was evaluated against cadmium induced hepatic cytotoxicity in culture and oxidative stress in rats. Nonmalignant "Chang" liver cell culture was exposed to Cd (cadmium chloride) that produced cytotoxicity in terms of increase in cell growth inhibition rate, alanine aminotransferase, lactate dehydrogenase and lipid peroxidation, which was significantly mitigated by pyridoxine in a concentration dependent manner. Acute exposure to Cd (6.

Protective effects of propolis on inorganic mercury induced oxidative stress in mice.

Protective potential of propolis was evaluated against mercury induced oxidative stress and antioxidant enzymatic alterations in mice liver. Exposure to mercuric chloride (HgCl2; 5 mg/kg; ip) induced oxidative stress by increasing lipid peroxidation and oxidized glutathione level along with concomitant decrease in glutathione and various antioxidant enzymes. Mercury intoxication deviated the activity of liver marker enzymes in serum.