Enhancing patient education in cancer care: Intelligent cancer patient education model for effective communication.

Background: The incidence of cancer is on the rise annually, whereas there exists a significant deficit of healthcare personnel. Inadequate communication between healthcare providers and patients may result in adverse emotional outcomes for the latter and interfere with their treatment progress. A viable solution to alleviate patient distress involves utilizing text generation models as an efficacious tool for delivering patient education.

STSRNet: Deep Joint Space-Time Super-Resolution for Vector Field Visualization.

We propose STSRNet, a joint space-time super-resolution deep learning based model for time-varying vector field data. Our method is designed to reconstruct high temporal resolution and high spatial resolution vector fields sequence from the corresponding low-resolution key frames. For large scale simulations, only data from a subset of time steps with reduced spatial resolution can be stored for post hoc analysis.

Helicobacter pylori inhibits autophagic flux and promotes its intracellular survival and colonization by down-regulating SIRT1.

Helicobacter pylori (H. pylori) is the strong risk factor for a series of gastric pathological changes. Persistent colonization of H.

SIRT1 inhibits chemoresistance and cancer stemness of gastric cancer by initiating an AMPK/FOXO3 positive feedback loop.

Chemotherapy is the standard care for patients with gastric cancer (GC); however, resistance to existing drugs has limited its success. The persistence of cancer stem cells (CSCs) is considered to be responsible for treatment failure. In this study, we demonstrated that SIRT1 expression was significantly downregulated in GC tissues, and that a low SIRT1 expression level indicated a poor prognosis in GC patients.

SIRT1 suppresses the migration and invasion of gastric cancer by regulating ARHGAP5 expression.

Gastric cancer (GC) ranks among the top five malignant tumors worldwide by the incidence and mortality rate. However, the mechanisms underlying its progression are poorly understood. In this study, we investigated the role of SIRT1, a class III deacetylase, in the invasion and metastasis of GC.