Publications by authors named "Yaojun Qin"

Auxin has emerged as a crucial regulator of plant nitrogen (N)-use efficiency (NUE) through indirect effects on plant growth and development and direct regulation of N metabolism-related genes. We previously reported DULL NITROGEN RESPONSE1 (DNR1) as an amino transferase that inhibits auxin accumulation and negatively regulates rice (Oryza sativa) NUE and grain yield. However, the identities of molecular regulators acting upstream of DNR1 await exploration.

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Between the two major rice subspecies, indica varieties generally exhibit higher nitrate (NO) uptake and nitrogen (N)-use efficiency (NUE) than japonica varieties. Introducing efficient NO utilization alleles from indica into japonica could improve NUE, and at the same time uncover unknown regulators of NO metabolism. Here, we identify OsWRKY23 as a key regulator of NO uptake and NUE differences between indica and japonica rice.

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Plant nitrogen (N)-use efficiency (NUE) is largely determined by the ability of root to take up external N sources, whose availability and distribution in turn trigger the modification of root system architecture (RSA) for N foraging. Therefore, improving N-responsive reshaping of RSA for optimal N absorption is a major target for developing crops with high NUE. In this study, we identified RNR10 (REGULATOR OF N-RESPONSIVE RSA ON CHROMOSOME 10) as the causal gene that underlies the significantly different root developmental plasticity in response to changes in N level exhibited by the indica (Xian) and japonica (Geng) subspecies of rice.

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The study aimed to synthesize novel zeolite substrates modified with four types of ZnAl-LDHs including Cl-LDHs(1:1), Cl-LDHs(3:1), CO-LDHs(1:1), and CO-LDHs(3:1); investigate Cr(VI) removal efficiencies in lab-scale constructed wetlands (CWs); and explore the effect of different Zn/Al ratios and intercalated anions on the removal efficiencies of Cr(VI) by modified zeolite. Different ZnAl-LDHs were prepared by co-precipitation method and coated onto the surface of original zeolite. Field emission scanning electron microscope and energy dispersive spectrometer were used to analyze physicochemical properties of zeolite/ZnAl-LDHs.

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Article Synopsis
  • Research showed that injecting apelin-13 into the brain significantly reduced pain responses in mice subjected to a pain test, indicating its potential antinociceptive effects.
  • The effects of apelin-13 were blocked by specific receptor antagonists, suggesting that both the APJ receptor and μ-opioid receptor are involved in its pain-relieving properties, and it can enhance the effectiveness of morphine.
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Article Synopsis
  • - Apelin-13, a peptide in the brain, was studied for its role in emotion-related behavior using tests like the forced swimming test (FST) and tail suspension test (TST), where it increased immobility time in a dose-dependent manner.
  • - The increase in immobility caused by apelin-13 was blocked by certain antagonists, indicating that its effects are mediated by the APJ receptor and κ-opioid receptor, but not by the corticotrophin-releasing factor receptor.
  • - Additional tests confirmed that apelin-13 did not disrupt spontaneous activity or motor function, suggesting that its influence on behavior is related specifically to inducing depression-like symptoms in the mice rather than affecting overall activity levels.*
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Apelin, the novel identified peptide, is the endogenous ligand for the APJ. Previous studies have reported the effect of apelin on food intake, however the action of acute central injected apelin on food intake in mice remains unknown. The present study was designed to investigate the mechanism as well as the effect of central apelin-13 on food intake in mice.

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Apelin, as the endogenous ligand for the APJ, regulates many biological functions, including blood pressure, neuroendocrine, drinking behavior, food intake and colonic motility. The present study was designed to investigate the effect of central apelin-13 on gastric emptying and gastrointestinal transit in mice. Intracerebroventricular (i.

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