Allelic variations of HMW-GS and LMW-GS and quality analysis in Yannong series wheat cultivars/derivative lines.

Introduction: Gluten quality is one of the most important traits of the common wheat ( L.). In Chinese wheat production, Yannong series cultivars/derivative lines possess unique characteristics and play an important role in both yield and quality contribution.

Characterization and identification of the powdery mildew resistance gene in wheat breeding line ShiCG15-009.

Powdery mildew, caused by Blumeria graminis f. sp. tritici (Bgt), is a serious fungal disease that critically threatens the yield and quality of wheat.

Identification of a Allele Conferring Powdery Mildew Resistance in Wheat Breeding Line GR18-1.

Powdery mildew caused by f. sp. () is a serious fungal wheat disease of wheat worldwide.

Identification of the powdery mildew resistance gene in wheat breeding line Yannong 99102-06188 bulked segregant exome capture sequencing.

Powdery mildew of wheat (), caused by f.sp. (), is a destructive disease that seriously threatens the yield and quality of its host.

Mining of Wheat Alleles for Goal-Oriented Marker-Assisted Breeding.

Powdery mildew of wheat, caused by f. sp. (), is a devastating disease that seriously reduces yield and quality worldwide.

The Fused/Smurf complex controls the fate of Drosophila germline stem cells by generating a gradient BMP response.

In the Drosophila ovary, germline stem cells (GSCs) are maintained primarily by bone morphogenetic protein (BMP) ligands produced by the stromal cells of the niche. This signaling represses GSC differentiation by blocking the transcription of the differentiation factor Bam. Remarkably, bam transcription begins only one cell diameter away from the GSC in the daughter cystoblasts (CBs).

Effects of estrogen and androgen deprivation on the progression of non-alcoholic steatohepatitis (NASH) in male Sprague-Dawley rats.

Aim: We studied the mechanisms of estrogen/androgen involvement in the induction of NASH by treating Sprague-Dawley (SD) rats fed with a normal or high fat (HF) diet by depriving them of endogenous estrogens/androgens.

Methods: Male adult SD rats (n = 10/group) on normal or HF diets were treated for 75 days either with tamoxifen (Tam) or flutamide (Flu) or Tam + Flu in order to induce NASH. We analyzed histopathologically the liver samples from the treated groups for NASH, checked the serum biochemical and lipid profile markers and finally analyzed the signal pathways underlying the molecular mechanisms for the induction process of NASH.