Publications by authors named "Y H Kuan"

Background: Although various repetitive transcranial magnetic stimulation (rTMS) and theta burst stimulation (TBS) protocols are used, their comparative effectiveness for treating poststroke hemineglect remains unassessed.

Objective: To investigate rTMS and TBS effects on clinical outcomes in poststroke hemineglect through a systematic review and network meta-analysis.

Methods: We searched PubMed, EMBASE, and Cochrane Library databases up to March 7, 2024, for trials on rTMS or TBS in poststroke hemineglect.

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  • Adrenocortical carcinoma (ACC) is a rare cancer originating from the adrenal cortex, with about half of the cases being functional and producing excess cortisol.
  • It is unusual for ACC to show high metanephrine levels, which are typically seen in pheochromocytoma.
  • A case study is presented of a large adrenal tumor displaying characteristics of both ACC and pheochromocytoma, ultimately diagnosed as metastatic ACC through biopsy.
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This meta-analysis investigated therapeautic effects of laser therapies in patients with Bell's palsy (BP). The authors performed the literature search in the PubMed, Embase, and Cochrane Library databases using the following search terms: (facial paralysis OR Bell's palsy OR facial palsy OR idiopathic facial paralysis) AND (laser OR low-level laser OR photobiomodulation OR phototherapy). Relevant studies published before October 29th 2024 were identified.

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  • Neurodegeneration and neuroinflammation play significant roles in Japanese Encephalitis virus (JEV) infection, with the NMDA receptor contributing to these processes through excitatory neurotoxicity and inflammation.
  • Research showed that JEV infection in rat microglia leads to glutamate release, activating harmful signaling pathways that result in pro-inflammatory responses and neurodegeneration.
  • NMDA receptor antagonists, such as MK801 and memantine, were found to reduce the harmful effects of JEV infection on microglia and neurons, indicating their potential as therapeutic options to mitigate neuroinflammation and cell death.
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