Publications by authors named "Xiurong Yan"
The biodegradable (polybutylene adipate terephthalate: PBAT) and conventional (polyethylene: PE) microplastics (MPs) at 0.5 %, 1 %, and 2 % dosages (w/w) were added into soils with and without Serratia marcescens ZY01 (ZY01, a tet-host strain) to understand their different effects on the dissipation of oxytetracycline (OTC) and tet. The results showed that the dosages of PBAT MP exhibited different inhibition degrees of OTC biodegradation in soils regardless of ZY01, while the dosages of PE MP did not change the enhancement degree of OTC biodegradation in soils without ZY01.
View Article and Find Full Text PDFArticle Synopsis
- The paper explores how biofilms colonize microplastics in aquatic environments, detailing bacterial traits and the environmental factors influencing biofilm formation.
- Biofilms provide microplastics with advantages like nutrient access and increased resistance to stress while altering their physical and chemical properties.
- The research emphasizes the complex interactions between biofilms and microplastics, highlighting their combined impact on pollutant behaviors and the accumulation of heavy metals, calling for more investigation into their environmental risks and management.
Sulfamethoxazole (SMX, a sulfonamide antibiotic) is ubiquitously present in various aqueous systems, which can accelerate the spread of antibiotic resistance genes, induce genetic mutations, and even disrupt the ecological equilibrium. Considering the potential eco-environmental risk of SMX, this study explored an effective technology using Shewanella oneidensis MR-1 (MR-1) and nanoscale zero-valent iron-enriched biochar (nZVI-HBC) to remove SMX from aqueous systems with different pollution levels (1-30 mg·L). SMX removal by nZVI-HBC and nZVI-HBC + MR-1 (55-100 %) under optimal conditions (iron/HBC ratio of 1:5, 4 g·L nZVI-HBC, and 10 % v/v MR-1) was more effective than its removal by MR-1 and biochar (HBC) (8-35 %).
View Article and Find Full Text PDFBackground: Local anesthetics in spinal anesthesia have neurotoxic effects, resulting in severe neurological complications. Intrathecal monosialoganglioside (GM1) administration has a therapeutic effect on bupivacaine-induced neurotoxicity. The aim of this study was to determine the underlying mechanisms of bupivacaine-induced neurotoxicity and the potential neuroprotective role of GM1.
View Article and Find Full Text PDFBackground: Bupivacaine causes neuronal and axonal degeneration, leading to cauda equina syndrome or permanent nerve damage. Our previous studies have shown that intrathecal or intravenous gangliosides monosialogangliosides (GM-1s) have therapeutic effects against bupivacaine-induced neurotoxicity, but we do not know what are the differences between the two methods.
Methods And Results: Bupivacaine-induced neurotoxicity was induced in rats by three times injection of 5% bupivacaine (0.