Role of Left-Hand Cut Contributions on Pole Extractions from Lattice Data: Case Study for T_{cc}(3875)^{+}.

We discuss recent lattice data for the T_{cc}(3875)^{+} state to stress, for the first time, a potentially strong impact of left-hand cuts from the one-pion exchange on the pole extraction for near-threshold exotic states. In particular, if the left-hand cut is located close to the two-particle threshold, which happens naturally in the DD^{*} system for the pion mass exceeding its physical value, the effective-range expansion is valid only in a very limited energy range up to the cut and as such is of little use to reliably extract the poles. Then, an accurate extraction of the pole locations requires the one-pion exchange to be implemented explicitly into the scattering amplitudes.

Understanding the 0 and 2 charmonium(-like) states near 3.9 GeV.

We propose that the X(3915) observed in the J/ψω channel is the same state as the χ(3930), and the X(3960), observed in the DD channel, is an S-wave DD hadronic molecule. In addition, the J=0 component in the B→DDK assigned to the X(3915) in the current Review of Particle Physics has the same origin as the X(3960), which has a mass around 3.94 GeV.

Prediction of a Narrow Exotic Hadronic State with Quantum Numbers J^{PC}=0^{--}.

Lots of charmonium-like structures have been observed in the last two decades. Most of them have quantum numbers that can be formed by a pair of charm and anticharm quarks, thus it is difficult to unambiguously identify the exotic ones among them. In this Letter, by exploiting heavy quark spin symmetry, we present a robust prediction of the hadronic molecular scenario, where the ψ(4230), ψ(4360) and ψ(4415) are identified as DD[over ¯]_{1}, D^{*}D[over ¯]_{1}, and D^{*}D[over ¯]_{2}^{*} bound states, respectively.

Is the existence of a J/ψJ/ψ bound state plausible?

In a recent measurement LHCb reported pronounced structures in the J/ψJ/ψ spectrum. One of the various possible explanations of those is that they emerge from non-perturbative interactions of vector charmonia. It is thus important to understand whether it is possible to form a bound state of two charmonia interacting through the exchange of gluons, which hadronise into two pions at the longest distance.

Erratum: Coupled-Channel Interpretation of the LHCb Double-J/ψ Spectrum and Hints of a New State Near the J/ψJ/ψ Threshold [Phys. Rev. Lett. 126, 132001 (2021)].

This corrects the article DOI: 10.1103/PhysRevLett.126.

Explaining the Many Threshold Structures in the Heavy-Quark Hadron Spectrum.

Tremendous progress has been made experimentally in the hadron spectrum containing heavy quarks in the last two decades. It is surprising that many resonant structures are around thresholds of a pair of heavy hadrons. There should be a threshold cusp at any S-wave threshold.

Coupled-Channel Interpretation of the LHCb Double-J/ψ Spectrum and Hints of a New State Near the J/ψJ/ψ Threshold.

Recently, the LHCb Collaboration reported pronounced structures in the invariant mass spectrum of J/ψ pairs produced in proton-proton collisions at the Large Hadron Collider. In this Letter, we argue that the data can be very well described within two variants of a coupled-channel approach employing T matrices consistent with unitarity: (i) with just two channels, J/ψJ/ψ and ψ(2S)J/ψ, as long as energy-dependent interactions in these channels are allowed, or (ii) with three channels J/ψJ/ψ, ψ(2S)J/ψ, and ψ(3770)J/ψ with just constant contact interactions. Both formulations hint at the existence of a near-threshold state in the J/ψJ/ψ system with the quantum numbers J^{PC}=0^{++} or 2^{++}, which we refer to as X(6200).

Regulation and mechanism of mouse miR-130a/b in metabolism-related inflammation.

Increasing evidence suggests that microRNAs are involved in regulating immune response and metabolism, which are among the most fundamental requirements for survival. Here we investigate the contribution and mechanism of microRNA-130a/b in controlling metabolism-related inflammation. Our findings indicate that miR-130a/b significantly inhibits TNFα and Sp1 expression by directly binding to their 3'-untranslated regions.