PbSO reaction mechanism in oxygen and reduction atmospheres during co-smelting process with primary lead material.

At present, lead-containing wastes have increasingly become the raw materials together with primary lead concentrate for lead production to meet the ever-increasing lead demand market. PbSO is the dominant component in the lead-containing wastes, nevertheless, its reaction behavior during lead smelting is not sufficiently investigated. This study investigated PbSO decomposition behaviors and phase transformation mechanisms at oxidizing and reductive atmospheres and various gas flow rates.

Microplastic types dominate the effects of bismuth oxide semi-conductor nanoparticles on their transport in saturated quartz sand.

The transport of microplastics (MPs) is of great significance due to its potential threat to subsurface systems. The copresence of MPs and semi-conductor nanoparticles is quite common in practical environments (i. e.

Mechanism of Sirtuin1-Mediated Deacetylation of p65-Mediated Ferroptosis of Hippocampal Neurons in Cerebral Injury after Cardiopulmonary Resuscitation in Rats.

Objective: Post-resuscitation brain injury is a common sequela after cardiac arrest (CA). Increasing sirtuin1 (SIRT1) has been involved in neuroprotection in oxygen-glucose deprivation (OGD) neurons, and we investigated its mechanism in post-cardiopulmonary resuscitation (CPR) rat brain injury by mediating p65 deacetylation modification to mediate hippocampal neuronal ferroptosis.

Methods: Sprague-Dawley rat CA/CPR model was established and treated with Ad-SIRT1 and Ad-GFP adenovirus vectors, or Erastin.

FTO rs1121980 polymorphism contributes to coronary artery disease susceptibility in a Chinese Han population.

Background: The fat mass and obesity-associated protein (FTO) has been showed to be involved in the pathogenesis and progression of coronary artery disease (CAD). However, the effects of FTO variants on CAD risk remain poorly understood. We herein genotyped three SNPs (rs1121980, rs72803657, and rs4783818) in FTO to investigate the influence of FTO polymorphisms on individual susceptibility to CAD.