Physiological, psychometric, and performance effects of the Christmas break in Australian football.

Purpose: The aim of the current study was to quantify the physiological, psychometric, and performance effects of a 2-wk Christmas break in a professional Australian Football League club.

Methods: A series of physiological (eg, heart-rate [HR] response to a 5-min submaximal run and skinfold thicknesses), psychometric (rating-of-perceived-exertion [RPE] responses and wellness variables), and performance (running activity during standardized handball games, isometric midthigh pull [IMTP] peak force, and countermovement jump [CMJ]) measures were conducted in the weeks before and after the break.

Results: There was a possible and small increase in the sum of 7 skinfolds, while body mass and fat-free mass remained possible and likely unchanged, respectively.

Influence of neuromuscular fatigue on accelerometer load in elite Australian football players.

Purpose: To determine the impact of neuromuscular fatigue (NMF) assessed from variables obtained during a countermovement jump on exercise intensity measured with triaxial accelerometers (load per minute [LPM]) and the association between LPM and measures of running activity in elite Australian Football.

Methods: Seventeen elite Australian Football players performed the Yo-Yo Intermittent Recovery Test level 2 (Yo-Yo IR2) and provided a baseline measure of NMF (flight time:contraction time [FT:CT]) from a countermovement jump before the season. Weekly samples of FT:CT, coaches' rating of performance (votes), LPM, and percent contribution of the 3 vectors from the accelerometers in addition to high-speed-running meters per minute at >15 km/h and total distance relative to playing time (m/min) from matches were collected.

Using eggs from Schistosoma mansoni as an in vivo model of helminth-induced lung inflammation.

Schistosoma parasites are blood flukes that infect an estimated 200 million people worldwide. In chronic infection with Schistosoma, the severe pathology, including liver fibrosis and splenomegaly, is caused by the immune response to the parasite eggs rather than the parasite itself. Parasite eggs induce a Th2 response characterized by the production of IL-4, IL-5 and IL-13, the alternative activation of macrophages and the recruitment of eosinophils.