Publications by authors named "Weldon H"

Objective: As surgical residency programs begin developing communication skills-specific training curricula, it is important to understand those skills required of surgeons in the many settings in which they work. It is also useful to observe the skills that residents already demonstrate, as well as those skills that need further development. This study evaluated surgical residents' communication behaviors across different services to understand how the clinical learning environment influences their approach to surgeon-patient interactions.

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Objective: An existing communication coaching program was adapted to address an educational gap to enhance the communication skills of surgical residents.

Design: Expert communication coaches observe surgical residents in various clinical settings and provide detailed feedback and recommendations for improvement.

Setting: Observations occur across diverse clinical environments, including inpatient and outpatient settings, peri-operative contexts, team meetings, and skills labs.

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While previous research has consistently found that negative forms of interparental conflict predict poorer outcomes in children, less is known about children's immediate responses to conflict. In a sample of 101 children (9-11 years of age) and their parents, we used a novel methodological approach to examine children's affect and perceived arousal responses to a live conflict between their parents in the lab. In addition, we examined children's self-reported cognitions regarding interparental conflict as predictors of these affect and perceived arousal responses.

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Bicyclic furanopyrimidines were recently discovered by us to be potent and selective inhibitors of VZV. Related studies to investigate the role of the sugar in this activity uncovered dideoxy furanopyrimidines as inhibitors of HCMV and this led to the preparation of highly modified long alkyl chain furanopyrimidines from the N- and O-alkylation of their parent bases. Herein we describe their synthesis and subsequent biological evaluation against HCMV.

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Glucosamine (Glmn), a product of glucose metabolism via the hexosamine pathway, causes insulin resistance in isolated adipocytes by impairing insulin-induced GLUT 4 glucose transporter translocation to the plasma membrane. We hypothesized that Glmn causes insulin resistance in vivo by a similar mechanism in skeletal muscle. We performed euglycemic hyperinsulinemic clamps (12 mU/kg/min + 3H-3-glucose) in awake male Sprague-Dawley rats with and without Glmn infusion at rates ranging from 0.

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