[Myoglobin release and renal function in polytraumatized patients in intensive care].

The excessive release of myoglobin following extensive skeletal muscle trauma, burns, and myopathies may result in renal dysfunction. Due to its molecular size, myoglobin is filtered through the glomerulus and is in part reabsorbed by the tubular system. intraluminal deposition of myoglobin following renal hypoperfusion and the impact of endogenous mediators on cell function contribute to the pathogenesis of acute renal failure.

[Energy demands and supply in polytraumatized patients in the postoperative phase].

Normal enteral feeding cannot be maintained after trauma. Post-aggression metabolism or socalled "flow phase" usually results from hormonal changes, including rise in catecholamine and cortisole, drop in testosterone as well as T3 and T4, and relative hGH deficit. Glucose intolerance, increased demand for energy, and catabolism are the consequences along with reduced protein uptake and increased protein breakdown with all associated complications.