Publications by authors named "W Jiao"
The combined application of dissimilatory iron-reducing bacteria (DIRB) and Fe(III) nanoparticles has garnered widespread interest in the contaminants transformation and removal. The efficiency of this composite system relies on the extracellular electron transfer (EET) process between DIRB and Fe(III) nanoparticles. While modifications to Fe(III) nanoparticles have demonstrated improvements in EET, enhancing DIRB activity also shows potential for further EET enhancement, meriting further investigation.
View Article and Find Full Text PDFPurpose: The objective of this study is to elucidate the sensitizing effect of mesoporous silica nanoparticles (MSNs) on shear wave elastography (SWE) and to investigate the potential application of MSNs as a sensitizer to enhance the sensitivity of SWE in the diagnosis of metabolic-associated steatohepatitis (MASH).
Materials And Methods: The in vitro gelatin models with varying ratios were assessed using SWE to identify the gelatin ratio that most closely approximates with human liver stiffness. Following the characterization of the dispersion properties of MSNs, in vitro models incorporating MSNs of different particle sizes were developed.
Background: Hemorrhage is the most common and dangerous complication after percutaneous nephrolithotripsy (PCNL). Therefore, this study introduces the management experience of bleeding complications in our center.
Methods: This retrospective study included 77 patients with severe bleeding after PCNL.
Article Synopsis
- The study investigates how interleukin-17 (IL-17) affects CD4+ T-cell immune regulation and the JAK/STAT signaling pathway, potentially leading to new therapies for dilated cardiomyopathy.
- Naive CD4+ T cells from mice were manipulated to either overexpress or knockdown IL-17, with multiple proteins measured via methods like Western blotting and PCR to assess inflammatory responses and signaling pathways.
- Results showed that overexpressing IL-17 increased levels of inflammatory factors and apoptosis in CD4+ T cells, while knocking it down decreased these levels, highlighting IL-17's significant regulatory role in inflammation and immune response.