Publications by authors named "W Chudzinski"
<b><br>Introduction:</b> Primary hyperparathyroidism (PHPT) is mainly caused by parathyroid adenoma (PA). Rare variants of PA, weighing >2.0-3.
View Article and Find Full Text PDFBACKGROUND Superior mesenteric artery (SMA) aneurysms account for about 5.5% of all visceral aneurysms, and are most commonly secondary to infectious causes or dissection. They tend to expand and rupture.
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- Parathyroid cystic adenomas (PCAs) are rare, making up 0.5-1% of parathyroid adenomas and contributing to 1-2% of primary hyperparathyroidism (PHPT) cases, prompting a study on their characteristics and associated risks.
- The study compared 17 patients with PHPT and parathyroid cysts to 100 patients with hyperparathyroidism due to other causes, focusing on various lab measurements before and after surgery.
- Findings indicated that patients with parathyroid cysts had significantly higher levels of PTH and calcium, suggesting they might be a unique risk factor for severe hypercalcemia, which can be potentially life-threatening.
BACKGROUND Brown and jaw tumors are rare entities of poorly understood etiology that are regarded as end-stage of bone remodeling in patients with long-lasting and chronic hyperparathyroidism. Jaw tumors are mainly diagnosed in jaw tumors syndrome (HPT-JT syndrome) and are caused by mutation in the CDC73 gene, encoding parafibromin, a tumor suppressing protein. The aim of this work is to present 4 cases of patients in whom the genetic mutation of the CDC73 gene and clinical presentation coexist in an unusual setting that has not yet been described.
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