Impact of Susceptibility on Plant Hormonal Composition during Clubroot Disease Development in Canola ().

Clubroot, caused by , is a soilborne disease of crucifers associated with the formation of large root galls. This root enlargement suggests modulation of plant hormonal networks by the pathogen, stimulating cell division and elongation and influencing host defense. We studied physiological changes in two cultivars, including plant hormone profiles-salicylic acid (SA), jasmonic acid (JA), abscisic acid (ABA), the auxin indole-3-acetic acid (IAA), and the ethylene precursor 1-aminocyclopropane-1-carboxylic acid (ACC)-along with their selected derivatives following inoculation with virulent and avirulent pathotypes.

Infection of canola by the root pathogen Plasmodiophora brassicae increases resistance to aboveground herbivory by bertha armyworm, Mamestra configurata Walker (Lepidoptera: Noctuidae).

Infection of plants by pathogens can result in the upregulation of induced defenses; plants may be more or less susceptible to attack by insect herbivores following infection. We investigated the interaction between canola, Brassica napus L., plants infected with clubroot, Plasmodiophora brassicae Woronin, and a generalist herbivore the bertha armyworm (BAW) Mamestra configurata Walker using two canola cultivars that varied in susceptibility to clubroot disease.

Virulence Spectrum of Single-Spore and Field Isolates of Able to Overcome Resistance in Canola ().

Clubroot, caused by Woronin, is an important disease of canola ( L.) that is managed mainly by planting clubroot-resistant (CR) cultivars. Field isolates of can be heterogeneous mixtures of various pathotypes, making assessments of the genetics of host-pathogen interactions challenging.