Publications by authors named "Vamsi A Alli"
Article Synopsis
- Trauma-hemorrhagic shock increases red blood cell (RBC) adhesion to endothelial cells, contributing to microvascular dysfunction and potential tissue injury.
- The study utilized both animal models and human blood samples to examine this phenomenon, focusing on the role of specific RBC surface adhesion molecules like CD36.
- Gut-derived factors in intestinal lymphatics were identified as significant triggers for these RBC changes, suggesting that manipulating this pathway might mitigate complications from trauma-hemorrhagic shock.
