Heterogeneous condensation in dense media.

A theoretical description of heterogeneous nucleation kinetics is presented. This description takes into account the perturbation of the vapor phase initiated by the growing droplets. The form of the density profile around the growing droplet is analyzed and some special approximations are given.

[Kallikrein-kinin system in acute empyema of pleura and pyopneumothorax].

In 56 patients with acute empyema of the pleura the level of aggregate kininogen and prekallikrein, free kallikrein and kininase activities in blood plasma and supernatant of pleural exudate were studied. It was established that epsilon-aminocapronic acid, contrykal and gordox suppress free kallikrein activity in the blood plasma and supernatant of pleural exudate. Intravenous and local application of proteolysis inhibitors resulted in improvement of the outcomes of the disease.

[Neuroendocrine regulation under maximal and subjectively optimal physical loads].

The responses of neuroendocrine system to maximum and subjective-optimum physical loading were found to be similar in young men and characterized by activation of adrenocortical, somatotropic and thyroid functions, particularly under maximum loading. The subjective-optimum loading was dosed out by the subjects themselves, differentiated individually and averaged 62% of the maximum oxygen consumption. The parameters of subjective-optimum physical loading were close to the threshold level necessary to stimulate neuroendocrine system and ensure an increase of somatotropine, cortisole, thyroxine and aldosterone concentration in the blood.

[Blood flow in the gastric mucosa and components of the kallikrein- kinin system in patients with duodenal ulcer].

The results of study of the blood flow rate in the gastric mucosa by means of intraoperative rheography and content of the main components of kallikrein-kinin system in the systemic and regional blood flow were compared. It was established that increase in the volumetric blood flow rate in patients with a complicated duodenal ulcer disease was caused by activation of the kallikrein-kinin system in the regional blood flow. Blockade of the celiac plexus has a pronounced effect on the state of a kallikrein-kinin system.