[Ultrastructure and metabolism of cardiomyocytes under experimental stress].

Morphogenesis of adaptational, disadaptational, and alterative changes affected structure and function of all live-supporting systems (contractile elements, macroergic phosphate synthesis, intracellular regeneration, and electrolyte exchange control) was studied in cardiomyocytes under experimental chronic stress using ultrastructural and cytochemical methods.

[The energy allowance and calcium homeostasis of the cardiomyocytes in the early stages of myocardial ischemia].

Morphofunctional equivalents are described of adaptive and abnormal reconstitution of CMC in acute coronary failure, using methods of light-transmission optics in combination with those of electron microscopy. It has been established that the contractile myocardium cell compensatory hyperfunction is provided by mobilization of a maximum number of the CMC organelles. Documented for the first time cytochemically is a direct interrelation between the metabolic activity of oxidoreductases, ability of mitochondria to accumulate Ca2+, and the status of their membranes.