Publications by authors named "V Flook"

This paper describes an attempt to calibrate a mathematical model that predicts the extent of bubble formation in both the tissue and blood of subjects experiencing decompression from a hyperbaric exposure. The model combines an inert gas dynamics model for uptake and elimination of inert anesthetic gases with a simple model of bubble dynamics in perfused tissues. The calibration has been carried out using the model prediction for volume of free gas (bubbles) as microl/ml in central venous blood and relating this to Doppler scores recorded at the end of hyperbaric exposures.

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An initial occupational survey (OS) was initiated to investigate the prevalence of venous gas embolism (VGE) in chamber attendants assisting hyperbaric oxygen (HBO2) treatments. Nine female subjects were exposed for three consecutive days to the routine hospital procedure of compressed air exposure to 240 kPa for approximately 115 min with 12 min of terminal oxygen (O2) breathing. VGE was monitored with ultrasound Doppler in 15 min intervals for 2h after the first and third exposure.

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Reduction in ascent speed and an increase in the O2 tension in the inspired air have been used to reduce the risk for decompression sickness. It has previously been reported that decompression speed and O2 partial pressure are linearly related for human decompressions from saturation hyperbaric exposures. The constant of proportionality K (K = rate/partial pressure of inspired O2) indicates the incidence of decompression sickness.

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Inspiratory dyspnea becomes an important factor in reducing a diver's ability to carry out physical work at depths in excess of 300 m. It is possible that dynamic compression of the trachea occurs when the intratracheal pressure drops below environmental pressure, thereby causing transient reduction in inspiratory flow. Vocal cords form an orifice of variable diameter, and orifice flow is predicted to occur at flow rates as low as 22 liter/min when gas density is 5 kg/m3 or more.

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