[Pathologic anatomy of Barrett's esophagus].

It is most substantiated to define Barrett's esophagus as intestinal metaplasia of esophageal cardiac mucosa irrespective of its association with the endoscopically detected esophageogastric junction, which develops as a result of gastroesophageal reflux. There is a need for further investigations of the specific features of the cardiac-type mucosa. During endoscopic study, it is important to be alert when identifying short and ultrashort Barrett's esophagus and to sample sufficient biopsy material.

[Ultrastructural and immunomorphological features of keratinisation in the inherited palmoplantar keratoderma and in the norm].

Disturbance of cell tonofibrillar apparatus plays a principal role in the development of PPK. This finally leads to inhibition of keratinisation and enhancement of cell proliferation. Change of the shape and size of keratohyalin granules are typical for all types of PPK.

[An electron radioautographic study of the vessels in the granulation tissue of long-term nonhealing wounds].

The wounds without healing for a long time (from 1 month to 2 years) were studied histologically and electron-radioautographically. The reduction of the vessel number in such wounds occurs, first, due to degenerative changes in the cells of their walls, and second, due to loss of connection between the cells. Some of these cells retain functional activity (incorporation of 3H-uridine) and capacity for proliferation (incorporation of 3H-thymidine).

[Electron microscope-radioautography study of inveterate non-healing wounds].

Morphological and functional peculiarities of long non-healing wound granulation tissue cells have been studied. The cell functional and proliferating activities were defined by electron-microscopic radioautography of 3H-uridine and 3H-thymidine incorporation. There are few vessels and the lumens are narrow.